首页> 外文会议>1996 Chinese Peptide Symposium July 21-25, 1996, Chengdu, China >CCK-B receptor activation in human lymphoblastic Jurkat cells results in AP-1 responsive genes activation
【24h】

CCK-B receptor activation in human lymphoblastic Jurkat cells results in AP-1 responsive genes activation

机译:人淋巴母细胞Jurkat细胞中的CCK-B受体激活导致AP-1响应基因激活

获取原文
获取原文并翻译 | 示例

摘要

1.Extracellular signals regulate many aspects of the growth and differentiation of animal cells. For example, receptors tyrosine kinases (RTK) and G-protein-coupled receptors (GPCR) which are among the various receptors located at the cell membrane are involved in the transfer of regulatory information from the cell surface to the nucleus. Once these receptors are stimulated, mitogen-activated protein kinases (MAPKs), also known as extracellular signal-regulated kinases (ERKs), can be stimulated and thus phosphorylate and regulate key intracellular enzymes and transcription factors involved i nthe control of cell proliferation as for example Fos and Jun [1].
机译:1,细胞外信号调节动物细胞生长和分化的许多方面。例如,位于细胞膜上的各种受体之中的受体酪氨酸激酶(RTK)和G蛋白偶联受体(GPCR)参与将调节信息从细胞表面转移至细胞核。一旦刺激了这些受体,就可以刺激有丝分裂原激活的蛋白激酶(MAPK),也称为细胞外信号调节激酶(ERK),从而磷酸化和调节关键的细胞内酶和转录因子,涉及细胞增殖的控制。例如Fos和Jun [1]。

著录项

相似文献

  • 外文文献
  • 中文文献
  • 专利
获取原文

客服邮箱:kefu@zhangqiaokeyan.com

京公网安备:11010802029741号 ICP备案号:京ICP备15016152号-6 六维联合信息科技 (北京) 有限公司©版权所有
  • 客服微信

  • 服务号