TENSION INDUCES CHANGES IN LIPID LATERAL DIFFUSION IN MODEL FLUID- PHASE MEMBRANES

机译：张力诱导模型相膜中脂类横向扩散的变化

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Shear stress due to blood flow on endothelial cells elicits numerous responses including G-protein coupled receptor activation and integrin-mediated signaling. Shear-induced change in membrane fluidity has been suggested to be one of the earliest mechanosensing mechanism involved in these processes [1, 2]. Alternatively, it has been suggested that shear forces are transduced through glycocalyx directly to transmembrane proteins and cytoskeleton [3], with very little shear force sensed by the membrane. It is not yet clear whether physiological tensions can alter membrane fluidity significantly.

机译：由于内皮细胞对血流引起的剪切应力引发了许多反应，包括G-蛋白偶联受体激活和整联蛋白介导的信号传导。已经提出了剪切诱导的膜流动变化是这些方法中涉及的最早机械沉积机制之一[1,2]。或者，已经提出，剪切力通过甘油癌直接转导给跨膜蛋白和细胞骨架[3]，由膜感测得非常小的剪切力。尚不清楚生理张力是否可以显着改变膜流动性。

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《ASME summer bioengineering conference;SBC2009》|2009年|P.1171-1172|共2页
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Hari S. Muddana; Ramachandra R. Gullapalli; Peter J. Butler;
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中图分类人体工程学;
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TENSION INDUCES CHANGES IN LIPID LATERAL DIFFUSION IN MODEL FLUID- PHASE MEMBRANES

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