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Maladaptive synaptogenesis induced by interaction of voltage gated calcium channel subunit alpha2delta-1 and thrombospondin-4 represents a new mechanism underlying neuropathic pain processing in nerve injury models.

机译：电压门控钙通道亚基α2delta-1和血小板反应蛋白-4相互作用引起的适应性突触形成是神经损伤模型中神经性疼痛处理的新机制。

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作者
Yu, Peter Yanhui.;
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作者单位

University of California, Irvine.;

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授予单位 University of California, Irvine.;
学科 Pharmacology.;Pacific Rim studies.
学位 Ph.D.
年度 2016
页码 95 p.
总页数 95
原文格式 PDF
正文语种 eng
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1. A novel mechanism of inhibition of high-voltage activated calcium channels by alpha-conotoxins contributes to relief of nerve injury-induced neuropathic pain. [J] . Klimis H, Adams DJ, Callaghan B, Pain. . 2011,第2期

机译：α-芋螺毒素抑制高压激活钙通道的新机制有助于缓解神经损伤引起的神经性疼痛。
2. ALTERED EXPRESSION OF THE VOLTAGE-GATED CALCIUM CHANNEL SUBUNIT alpha(2)delta-1: A COMPARISON BETWEEN TWO EXPERIMENTAL MODELS OF EPILEPSY AND A SENSORY NERVE LIGATION MODEL OF NEUROPATHIC PAIN [J] . Nieto-Rostro M., Sandhu G., Bauer C. S., Neuroscience: An International Journal under the Editorial Direction of IBRO . 2014,第Null期

机译：电压控制钙通道亚基α（2）delta-1的改变表达：两种实验性癫痫实验模型与神经痛疼痛感神经连接模型的比较
3. Allosteric interactions between receptor site 3 and 4 of voltage-gated sodium channels: a novel perspective for the underlying mechanism of scorpion sting-induced pain [J] . Yi-Jun Feng, Qi Feng, Jie Tao, Journal of Venomous Animals and Toxins including Tropical Diseases . 2015,第1期

机译：电压门控钠通道的受体位点3和4之间的变构相互作用：蝎刺引起疼痛的潜在机制的新观点
4. Post-transcriptional Regulation of Voltage-Gated Sodium Channel 1.8 (Nav1.8): A Key Underlying Mechanism of Neuropathic Pain Pathogenesis. [D] . Ruangsri, Supanigar. 2010

机译：电压门控钠通道1.8（Nav1.8）的转录后调控：神经性疼痛发病机制的关键基础机制。
5. Altered expression of the voltage-gated calcium channel subunit α2δ-1: A comparison between two experimental models of epilepsy and a sensory nerve ligation model of neuropathic pain [O] . M. Nieto-Rostro, G. Sandhu, C.S. Bauer, -1

机译：电压门控钙通道亚基α2δ-1的表达改变：两种癫痫实验模型与神经性疼痛的感觉神经结扎模型之间的比较
6. A novel mechanism of inhibition of high-voltage activated calcium channels by α-conotoxins contributes to relief of nerve injury-induced neuropathic pain [O] . Harry Klimis, D. J. Adams, B. Callaghan, 2011

机译：α-蓬松毒素的高压活化钙通道抑制的新机制有助于缓解神经损伤诱导的神经性疼痛

Maladaptive synaptogenesis induced by interaction of voltage gated calcium channel subunit alpha2delta-1 and thrombospondin-4 represents a new mechanism underlying neuropathic pain processing in nerve injury models.

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