被侵权的LV

丹棱

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被侵权的LV

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知识产权故事:当LV成为方向盘套的商标提起LV(路易威登),几乎无人不知. 早在1985年,品牌持有人路易威登就向商标局提出了注册申请,并在第18类皮革和人造皮革及制品上获得了注册商标的专用权.自1992年进入中国市场以来,LV已经在全国30多座城市开设了50多家门店.昂贵的价格、高端的品质,LV俨然是真正"高大上"的代言.

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《法律与生活》 |2015年第22期|42-44|共3页
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丹棱;
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1. LV起诉日本独立品牌商侵权胜诉 [J] . . 电子知识产权 . 2018,第11期
2. LV商标侵权案落槌 [J] . . 中国防伪报道 . 2009,第002期
3. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
4. 中国侵权行为形态与侵权责任形态法律适用指引——中国侵权责任法重述之侵权行为形态与侵权责任形态 [J] . 杨立新 . 河南财经政法大学学报 . 2013,第005期
5. 中国侵权行为形态与侵权责任形态法律适用指引——中国侵权责任法重述之侵权行为形态与侵权责任形态 [J] . 杨立新 . 河南财经政法大学学报 . 2013,第005期
6. 电子商务平台服务提供者商标间接侵权责任研究——论侵权责任法第三十六条在电子商务商标侵权中的适用 [C] . 司晓 ,费兰芳 . 2012知识产权南湖论坛暨“知识产权：制度完善与产业发展”国际研讨会 . 2012
7. 论商标的合理使用——以VOLVO诉瑞安公司商标侵权案为例 [A] . 王佳瑜 . 2009

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