• 主题
高级搜索  >
历史搜索 清空历史
首页> 美国卫生研究院文献>G3: GenesGenomesGenetics >Pollen Killer Gene S35 Function Requires Interaction with an Activator That Maps Close to S24 Another Pollen Killer Gene in Rice
【2h】

Pollen Killer Gene S35 Function Requires Interaction with an Activator That Maps Close to S24 Another Pollen Killer Gene in Rice

机译：花粉杀手基因S35功能需要与一个与S24（水稻中另一个花粉杀手基因）接近的激活剂相互作用

代理获取
本网站仅为用户提供外文OA文献查询和代理获取服务，本网站没有原文。下单后我们将采用程序或人工为您竭诚获取高质量的原文，但由于OA文献来源多样且变更频繁，仍可能出现获取不到、文献不完整或与标题不符等情况，如果获取不到我们将提供退款服务。请知悉。
页面导航
  • 摘要
  • 著录项
  • 相似文献
  • 相关主题

摘要

Pollen killer genes disable noncarrier pollens, and are responsible for male sterility and segregation distortion in hybrid populations of distantly related plant species. The genetic networks and the molecular mechanisms underlying the pollen killer system remain largely unknown. Two pollen killer genes, S24 and S35, have been found in an intersubspecific cross of Oryza sativa ssp. indica and japonica. The effect of S24 is counteracted by an unlinked locus EFS. Additionally, S35 has been proposed to interact with S24 to induce pollen sterility. These genetic interactions are suggestive of a single S24-centric genetic pathway (EFS–S24–S35) for the pollen killer system. To examine this hypothetical genetic pathway, the S35 and the S24 regions were further characterized and genetically dissected in this study. Our results indicated that S35 causes pollen sterility independently of both the EFS and S24 genes, but is dependent on a novel gene close to the S24 locus, named incentive for killing pollen (INK). We confirmed the phenotypic effect of the INK gene separately from the S24 gene, and identified the INK locus within an interval of less than 0.6 Mb on rice chromosome 5. This study characterized the genetic effect of the two independent genetic pathways of INK–S35 and EFS–S24 in indica–japonica hybrid progeny. Our results provide clear evidence that hybrid male sterility in rice is caused by several pollen killer networks with multiple factors positively and negatively regulating pollen killer genes.
机译：花粉杀手基因使非携带者花粉失去功能，并导致远缘相关植物物种的杂交种群中的雄性不育和分离畸变。花粉杀手系统背后的遗传网络和分子机制仍然未知。在稻的亚种间杂交中发现了两个花粉杀手基因S24和S35。 and和粳稻。 S24的作用被未连锁的基因座EFS抵消。另外，已经提出S35与S24相互作用以诱导花粉无菌。这些遗传相互作用提示花粉杀手系统有一条以S24为中心的遗传途径（EFS–S24–S35）。为了检验这种假设的遗传途径，在本研究中进一步鉴定了S35和S24区域并进行了遗传解剖。我们的结果表明，S35导致花粉不育，与EFS和S24基因无关，但依赖于接近 S24 基因座的新基因，该基因名为杀死花粉的诱因（ 墨水）。我们证实了 INK 基因的表型效应与 S24 基因分开，并在小于0.6 Mb的间隔内鉴定了 INK 基因座。水稻5号染色体。本研究描述了 INK–S35 和 EFS–S24 两个独立的遗传途径对 ja-粳杂种的遗传效应后代。我们的结果提供了明确的证据，表明水稻中的杂交雄性不育是由几种花粉杀手网络引起的，这些网络具有多个正负调控花粉杀手基因的因素。

著录项

相似文献

  • 外文文献
  • 中文文献
  • 专利
代理获取

客服邮箱：kefu@zhangqiaokeyan.com

京公网安备：11010802029741号 ICP备案号：京ICP备15016152号-6 六维联合信息科技 (北京) 有限公司©版权所有

  • 客服微信

  • 服务号