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首页> 外文期刊>World Journal of Gastroenterology >Pseudoachalasia in a patient after truncal vagotomy surgery successfully treated by subsequent pneumatic dilations.
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Pseudoachalasia in a patient after truncal vagotomy surgery successfully treated by subsequent pneumatic dilations.

机译：截骨迷走神经切断术术后患者的假性ach门失弛缓症通过随后的气道扩张术成功治疗。

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摘要

Pseudoachalasia is a difficult condition for the clinician to differentiate from idiopathic achalasia even by manometry, radiological studies or endoscopy. Its etiology is usually associated with tumors. In most cases, the diagnosis is made after surgical explorations. The proposed pathogenesis of the disease is considered as mechanical obstruction of the distal esophagus or infiltration of the malignancy that affects the inhibitory neurons of the meyenteric plexus in the majority of cases. Surgery has been reported as a cause of pseudoachalasia. We report a 70-year-old man who suffered from deglutination disorder caused by pseudo-achalasia after truncal vagotomy. The patient was symptom-free after a nine-year follow-up and complete recovery of esophageal motility status from pseudoachalasia after pneumatic dilations. We also reviewed the literature of pseudoachalasia.
机译：即使通过测压，放射学检查或内窥镜检查，假性ach门失弛缓症对于临床医生来说也很难与特发性门失弛缓症区分开。其病因通常与肿瘤有关。在大多数情况下，诊断是在手术探查之后做出的。在大多数情况下，这种疾病的发病机制被认为是食管远端的机械性阻塞或恶性肿瘤的浸润，从而影响了肠系膜神经丛的抑制性神经元。据报道手术是假性ach门失弛缓的原因。我们报告了一名70岁的男子，他因截断迷走神经切断术而遭受了由假性asia门失弛缓症引起的脱水现象。经过九年的随访，患者无症状，并在气管扩张后从假性ach门失弛缓症完全恢复了食管的蠕动状态。我们还回顾了假性al门失弛缓症的文献。

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