• 主题
高级搜索  >
历史搜索 清空历史
首页> 外文期刊>World Journal of Gastroenterology >Smoking is not associated with nonalcoholic fatty liver disease.
【24h】

Smoking is not associated with nonalcoholic fatty liver disease.

机译：吸烟与非酒精性脂肪肝无关。

获取原文
获取原文并翻译 | 示例
           
页面导航
  • 摘要
  • 著录项
  • 相似文献
  • 相关主题

摘要

AIM: To analyze the relationship between smoking and nonalcoholic fatty liver disease (NAFLD). METHODS: This is a cross-sectional study of a healthy population, carried out in a check-up unit of a university hospital in Mexico City. We enrolled 933 subjects, 368 current smokers (cases) and 565 persons who had never smoked (controls). Demographic, metabolic and biochemical variables were measured in the two groups. NAFLD was determined by ultrasound and metabolic syndrome according to ATPIII. RESULTS: A total of 548 men (205 cases and 343 controls) and 337 women (114 cases and 223 controls) were included in the analysis. Statistical differences between cases and controls were observed only in high blood pressure prevalence (6.6% vs 11.3%, P < 0.05; cases and controls respectively), high-density lipoproteins (1.00 +/- 0.26 vs 1.06 +/- 0.28 mmol/L, P < 0.005), triglycerides (2.18 +/- 1.49 vs 1.84 +/- 1.1 mmol/L, P < 0.001), and erythrocyte sedimentation rate (11.3 +/- 9.3 vs 13.5 +/- 11.9 mm/h, P < 0.001). No differences were observed in the prevalence of NAFLD (22.27% vs 29.68%, P = NS) and metabolic syndrome (41.69% vs 36.74%, P = NS). Univariate analysis showed that smoking was not a risk factor for NAFLD (OR = 0.89, 95% CI 0.65-1.21). CONCLUSION: No differences in NAFLD prevalence were observed between current smokers and nonsmokers, and furthermore, no differences were observed in heavy smokers (more than 20 packs/year), indicating that there is no relationship between smoking and NAFLD.
机译：目的：分析吸烟与非酒精性脂肪肝（NAFLD）之间的关系。方法：这是对健康人群的横断面研究，是在墨西哥城一家大学医院的检查部门中进行的。我们招募了933名受试者，368名当前吸烟者（病例）和565名从未吸烟的人（对照组）。在两组中测量了人口，代谢和生化变量。根据ATPIII，通过超声和​​代谢综合征确定了NAFLD。结果：总共包括548名男性（205例和343名对照）和337名女性（114例和223名对照）进行了分析。仅在高血压患病率（6.6％vs 11.3％，P <0.05；分别为病例和对照），高密度脂蛋白（1.00 +/- 0.26 vs 1.06 +/- 0.28 mmol / L）上观察到病例和对照之间的统计学差异。 ，P <0.005），甘油三酸酯（2.18 +/- 1.49与1.84 +/- 1.1 mmol / L，P <0.001）和红细胞沉降速率（11.3 +/- 9.3与13.5 +/- 11.9 mm / h，P < 0.001）。 NAFLD的患病率（22.27％vs 29.68％，P = NS）和代谢综合征（41.69％vs 36.74％，P = NS）没有差异。单因素分析表明，吸烟不是NAFLD的危险因素（OR = 0.89，95％CI 0.65-1.21）。结论：目前的吸烟者与不吸烟者之间，NAFLD患病率没有差异，此外，大量吸烟者（每年超过20包）中也未观察到差异，这表明吸烟与NAFLD之间没有关系。

著录项

相似文献

  • 外文文献
  • 中文文献
  • 专利
获取原文

客服邮箱：kefu@zhangqiaokeyan.com

京公网安备：11010802029741号 ICP备案号：京ICP备15016152号-6 六维联合信息科技 (北京) 有限公司©版权所有

  • 客服微信

  • 服务号