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首页> 外文期刊>World Journal of Gastroenterology >Relationship between lymphocyte apoptosis and endotoxin translocation after thermal injury in rats
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Relationship between lymphocyte apoptosis and endotoxin translocation after thermal injury in rats

机译：大鼠热损伤后淋巴细胞凋亡与内毒素易位的关系

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CONCLUSION: Gut-origin LPS is a major cause of endotoxemia taken place early in rats following severe thermal injury and could induce extensive lymphocyte apoptosis in blood and MLN, which suggests an immunosuppression state could follow the initial injury and favores a septic state based on apoptotic mechanism. AIM: To investigate the relationship between lymphocyte apoptosis in peripheral blood, spleen and mesenteric lymph nodes(MLN) and endotoxin translocation after thermal injury in rats. METHODS: In a Wistar rat model inflicted with 30/100 TBSA Ⅲ degree scalding, serum LPS levels in portal vein and vena cava were quantified by tachypleus amebocyte lysate (TAL) technique.
机译：结论：原肠脂多糖是严重热损伤后大鼠早期发生内毒素血症的主要原因，可诱导血液和MLN中广泛的淋巴细胞凋亡，这表明免疫抑制状态可跟随初始损伤，并倾向于基于凋亡的败血病状态。机制。目的：探讨热损伤后外周血，脾脏和肠系膜淋巴结（MLN）淋巴细胞凋亡与内毒素易位的关系。方法：在Wistar大鼠30/100 TBSAⅢ度烫伤模型中，通过速动性阿贝细胞溶解物（TAL）技术定量测定门静脉和腔静脉中的血清LPS水平。

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