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首页> 外文期刊>Case Reports in Obstetrics and Gynecology >Extreme Maternal Metabolic Acidosis Leading to Fetal Distress and Emergency Caesarean Section
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Extreme Maternal Metabolic Acidosis Leading to Fetal Distress and Emergency Caesarean Section

机译:极端的母亲代谢性酸中毒导致胎儿窘迫和紧急剖腹产

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A 31-year-old pregnant woman (32+3weeks) was admitted with extreme tachypnea. She had a previous history of congenital muscular dystrophy (Ullrich’s disease) and isolated glucosuria. The patient had reduced food intake during the last 24 hours prior to admission and vomited twice. Serum glucose level was normal (112 mg/dL), while urinalysis revealed glucosuria 4+ and ketonuria 4+. ABG revealed pH 7.06, PCO29 mm Hg, and bicarbonate 2 mmol/L. Anion gap was 28 mmol/L. Tachypnea was a compensatory mechanism for a severe nonlactic metabolic acidosis. The diagnosis of starvation ketoacidosis was established. The patient received supplemental dextrose 10% intravenously and sodium bicarbonate. As fetal heart monitoring was pathological, an emergency caesarean section was performed. Umbilical cord venous pH was 7.01, with PCO234 mm Hg and bicarbonate 8 mmol/L. Starvation ketoacidosis is a rare metabolic disorder that may occur mainly in the third trimester of pregnancy. Muscular dystrophy and renal glucosuria were precipitating factors.
机译:一名31岁的孕妇(32 + 3周)被接纳患有极速呼吸。她曾有先天性肌营养不良症(乌尔里希氏病)和孤立的糖尿病史。患者入院前24小时内食物摄入减少,呕吐两次。血清葡萄糖水平正常(112 mg / dL),而尿液分析显示糖尿4+和酮尿4+。 ABG的pH值为7.06,PCO29 mm Hg,碳酸氢盐为2 mmol / L。阴离子间隙为28 mmol / L。呼吸急促是严重的非乳酸代谢性酸中毒的一种补偿机制。建立了饥饿性酮症酸中毒的诊断。患者静脉补充10%葡萄糖和碳酸氢钠。由于胎儿心脏监护是病理性的,因此进行了紧急剖腹产。脐带静脉pH为7.01,PCO234 mm Hg,碳酸氢根为8 mmol / L。饥饿酮症酸中毒是一种罕见的代谢紊乱,可能主要发生在妊娠晚期。肌肉营养不良和肾糖尿症是诱发因素。

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