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The possible role of meditation in myofascial pain syndrome: A new hypothesis

机译：冥想在肌筋膜疼痛综合征中的可能作用：新假设

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Background of Hypothesis: Myofascial pain syndrome (MPS) is the most common musculoskeletal pain disorder of the head and neck area. In the past, several theories were put forth to explain its origin and nature, but none proved complete. Myofascial pain responds to changing psychological states and stress, anxiety, lack of sleep, anger, depression and chronic pain are direct contributional factors. Myofascial pain syndrome may be considered as a psychosomatic disorder. There are numerous accepted palliative approaches, but of all, relaxation techniques stand out and initiate healing at the base level. In this article, the connection between mental factors, MPS and meditation are highlighted. Recent literature has shed light on the fundamental role of free radicals in the emergence of myofascial pain. The accumulating free radicals disrupt mitochondrial integrity and function, leading to sustenance and progression of MPS. Meditation on the other hand was shown to reduce free radical load and can result in clinical improvement. 'Mindfulness' is the working principle behind the effect of all meditations, and I emphasize that it can serve as a potential tool to reverse the neuro-architectural, neurobiological and cellular changes that occur in MPS. Conclusions: The findings described in this paper were drawn from studies on myofascial pain, fibromyalgia, similar chronic pain models and most importantly from self experience (experimentation). Till date, no hypothesis is available connecting MPS and meditation. Mechanisms linking MPS and meditation were identified, and this paper can ignite novel research in this direction.
机译：假设的背景：肌筋膜疼痛综合征（MPS）是头颈部区域最常见的肌肉骨骼疼痛症。过去，提出了几种理论来解释其起源和性质，但没有一个被证明是完整的。肌筋膜疼痛对变化的心理状态做出反应，压力，焦虑，睡眠不足，愤怒，抑郁和慢性疼痛是直接的促成因素。肌筋膜疼痛综合征可被视为心身疾病。有许多公认的姑息治疗方法，但最重要的是，放松技术脱颖而出，并在基础水平上开始治愈。在本文中，强调了心理因素，MPS和冥想之间的联系。最近的文献阐明了自由基在肌筋膜疼痛出现中的基本作用。积累的自由基破坏线粒体的完整性和功能，导致MPS的维持和发展。另一方面，冥想被证明可以减少自由基负荷，并可以改善临床状况。 “正念”是所有冥想作用背后的工作原理，我强调它可以作为逆转MPS中发生的神经结构，神经生物学和细胞变化的潜在工具。结论：本文描述的发现来自肌筋膜疼痛，纤维肌痛，类似的慢性疼痛模型的研究，最重要的是来自自我经验（实验）。到目前为止，尚无关于将MPS和冥想联系起来的假设。确定了MPS和冥想之间的联系机制，可以激发这一方向的新颖研究。

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