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首页> 外文期刊>The Open Obesity Journal >Contributors to Pediatric Obesity in Adolescence: More than just Energy Imbalance
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Contributors to Pediatric Obesity in Adolescence: More than just Energy Imbalance

机译：青少年肥胖症的成因：不仅仅是能量失衡

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Disentangling the etiology of pediatric obesity continues to challenge researchers. Due to rapid growth anddevelopment, changes in the hormonal milieu, increased autonomy in feeding practices and greater interactions withenvironmental factors, adolescence is a particularly important period for the determination of body compositiontrajectories and the relationship to current and future obesity outcomes. A plethora of studies have focused on excessenergy consumption and physical inactivity as they relate to weight and fat gain in adolescence. Although these “BigTwo” have an impact, the increasing trends in pediatric obesity are not accounted for solely by increased energy intakeand decreased physical activity. Indeed, under similar conditions of energy balance, inter-individual variation in fataccumulation has been consistently noted. It is becoming more evident that additional factors may contributeindependently and/or synergistically to the increase in obesity. Such factors include (but are not limited to) metabolicprogramming in utero and in early childhood, the hormonal environment, endocrine disruptors, parental feeding practices,and the built environment. Our objective, therefore, is to investigate possible factors, particularly in adolescence thatcontributes to the increase in pediatric obesity beyond “The Big Two”.
机译：解开小儿肥胖的病因继续挑战研究人员。由于快速的生长发育，荷尔蒙环境的变化，进食方式的自主性增加以及与环境因素的更多相互作用，青春期是确定身体成分轨迹以及与当前和未来肥胖结局关系的特别重要的时期。大量的研究都集中在能量消耗过多和缺乏运动方面，因为它们与青春期的体重和脂肪增加有关。尽管这些“ BigTwo”产生了影响，但儿童肥胖症的增加趋势并不能仅通过增加能量摄入和减少体育锻炼来解决。的确，在相似的能量平衡条件下，脂肪累积的个体差异一直得到关注。越来越明显的是，其他因素可能独立地和/或协同地促进肥胖症的增加。这些因素包括（但不限于）子宫和幼儿期的代谢程序，激素环境，内分泌干扰物，父母的进食习惯和室内环境。因此，我们的目标是研究可能的因素，尤其是青春期导致“两大肥胖”以外的儿童肥胖增加的因素。

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