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首页> 外文期刊>Obesity facts : the European journal of obesity. >Hypocaloric Diet and Regular Moderate Aerobic Exercise Is an Effective Strategy to Reduce Anthropometric Parameters and Oxidative Stress in Obese Patients
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Hypocaloric Diet and Regular Moderate Aerobic Exercise Is an Effective Strategy to Reduce Anthropometric Parameters and Oxidative Stress in Obese Patients

机译：低毒性饮食和常规适度的有氧运动是一种有效的策略，以减少肥胖患者中的人体测量参数和氧化应激

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Background: Studies show that diet and exercise are important in the treatment of obesity. The aim of this study was to determine whether additional regular moderate aerobic exercise during a treatment with hypocaloric diet has a beneficial effect on oxidative stress and molecular damage in the obese patient. Methods: Oxidative stress of 16 normal-weight (NW) and 32 obese 1 (O1) subjects (BMI 30–34.9 kg/m2) were established by biomarkers of oxidative stress in plasma. Recombinant human insulin was incubated with blood from NW or O1 subjects, and the molecular damage to the hormone was analyzed. Two groups of treatment, hypocaloric diet (HD) and hypocaloric diet plus regular moderate aerobic exercise (HDMAE), were formed, and their effects in obese subjects were analyzed. Results: The data showed the presence of oxidative stress in O1 subjects. Molecular damage and polymerization of insulin was observed more frequently in the blood from O1 subjects. The treatment of O1 subjects with HD decreased the anthropometric parameters as well as oxidative stress and molecular damage, which was more effectively prevented by the treatment with HDMAE. Conclusion: HD and HDMAE treatments decreased anthropometric parameters, oxidative stress, and molecular damage in O1 subjects.
机译：背景：研究表明，饮食和运动在治疗肥胖症中是重要的。本研究的目的是判断患有低核饮食治疗过程中的额外常规适度的有氧运动对肥胖患者的氧化应激和分子损伤有益。方法：通过血浆中氧化应激的生物标志物建立16个正常重量（NW）和32个肥胖1（o1）对象（BMI 30-34.9kg / m 2）的氧化应激。将重组人胰岛素与来自NW或O1受试者的血液一起孵育，分析了对激素的分子损伤。形成了两组治疗，低匹配饮食（HD）和低核饮食加上常规适度的有氧运动（HDMAE），分析了它们对肥胖受试者的影响。结果：数据显示O1受试者中存在氧化应激。从O1受试者中更频繁地观察胰岛素的分子损伤和聚合。 HD的O1受试者的治疗降低了人体测量参数以及氧化应激和分子损伤，通过HDMAE处理更有效地防止。结论：HD和HDMAE治疗降低了O1受试者的人体测量参数，氧化应激和分子损伤。

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