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首页> 外文期刊>Journal of International Medical Research >Pathogenesis of traumatic temporomandibular joint ankylosis: a narrative review
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Pathogenesis of traumatic temporomandibular joint ankylosis: a narrative review

机译：创伤性颞下颌关节关节症的发病机制：叙事综述

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Objective To comprehensively review the literature and summarize the results from human and animal studies related to the possible causes and pathogenesis of traumatic temporomandibular joint ankylosis (TMJA). Materials and Methods The Google Scholar, Embase, and Web of Science databases were used to search for articles related to traumatic TMJA from 2011 to 2020. All articles were screened according to the inclusion and exclusion criteria, collected, and analyzed. Results Nineteen relevant articles were collected. These articles were classified into three groups: predisposing and etiological factors, cellular studies, and molecular studies. Conclusion The pathological mechanisms are similar between TMJA and nonunion hypertrophy. Aberrant structural and etiological factors as well as disordered cellular and molecular mechanisms might contribute to TMJA formation. Although preclinical and clinical data have provided new evidence on the pathogenesis of traumatic TMJA, the molecular mechanisms and biological events require further exploration.
机译：目的综合审查文献，总结了人类和动物研究的结果，与创伤性颞下颌关节骨质症（TMJA）的可能原因和发病机制有关。材料和方法用于从2011到2020年搜索与创伤TMJA相关的文章，从2011年到2020年搜索相关的文章。根据包含和排除标准，收集和分析，所有物品都被筛选。结果收集了20.1篇相关文章。这些物品被分为三组：易感和病因因素，细胞研究和分子研究。结论TMJA和壬疮肥大之间的病理机制相似。异常结构和病因因子以及无序的细胞和分子机制可能有助于TMJA形成。虽然临床前和临床资料已经为创伤性TMJA发病机制提供了新的证据，但分子机制和生物事件需要进一步的探索。

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