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首页> 外文期刊>Environmental Science & Technology >Association of Cardiopulmonary Health Effects with Source-Appointed Ambient Fine Particulate in Beijing, China: A Combined Analysis from the Healthy Volunteer Natural Relocation (HVNR) Study
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Association of Cardiopulmonary Health Effects with Source-Appointed Ambient Fine Particulate in Beijing, China: A Combined Analysis from the Healthy Volunteer Natural Relocation (HVNR) Study

机译：中国北京心肺健康影响与源指定环境微粒的关联：健康志愿者自然重定位（HVNR）研究的综合分析

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摘要

Previous studies have associated ambient particulate chemical constituents with adverse cardiopulmonary health effects. However, specific pollution sources behind the cardiopulmonary health effects of ambient particles are uncertain. We examined the cardiopulmonary health effects of fine particles (PM_(2.5)) from different pollution sources in Beijing, China, among a panel of 40 healthy university students. Study subjects were repeatedly examined for a series of cardiopulmonary health indicators during three 2-month-long study periods (suburban period, urban period 1, and urban period 2) in 2010-2011 before and after relocating from a suburban campus to an urban campus with changing air pollution levels and contents. Daily ambient PM_(2.5) mass samples were collected over the study and measured for 29 chemical constituents in the laboratory. Source appointment for ambient PM_(2.5) was performed using Positive Matrix Factorization, and mixed-effects models were used to estimate the cardiopulmonary effects associated with source-specific PM_(2.5) concentrations. Seven PM_(2.5) sources were identified as traffic emissions (12.0%), coal combustion (22.0%), secondary sulfateitrate (30.2%), metallurgical emission (0.4%), dust/soil (12.4%), industry (6.9%), and secondary organic aerosol (9.9%). Ambient PM_(2.5) in the suburban campus had larger contributions from secondary sulfateitrate (41.8% vs. 22.9%-26.0%) and metallurgical emission (0.7% vs. 0.3%) as compared to that in the urban campus), whereas PM_(2.5) in the urban campus had larger contributions from traffic emissions (13.0%-16.3% vs. 5.1%), coal combustion (21.0%-30.7% vs. 10.7%), and secondary organic aerosol (9.7%-12.0% vs. 8.7%) as compared to that in the suburban campus. Potential key sources were identified for PM_(2.5) effects on inflammatory biomarkers (secondary sulfateitrate and dust/soil), blood pressure (coal combustion and metallurgical emission), and pulmonary function (dust/soil and industry). Analyses using another source appointment tool Unmix yielded a similar pattern of source contributions and associated health effects. In conclusion, ambient PM_(2.5) in Beijing suburban and urban areas has two distinct patterns of source contributions, and PM_(2.5) from different sources may play important roles on different aspects of PM_(2.5)-reIated cardiopulmonary health effects.
机译：先前的研究已经将周围的颗粒化学成分与不利的心肺健康影响相关联。然而，环境颗粒物对心肺健康影响背后的特定污染源尚不确定。我们在来自40名健康大学生的小组中检查了来自中国北京不同污染源的细颗粒（PM_（2.5））对心肺健康的影响。在从郊区校园搬迁到城市校园之前和之后的2010年至2011年的三个为期两个月的研究期（郊区，城市时期1和城市时期2）中，反复对研究对象进行了一系列心肺健康指标的检查随着空气污染水平和含量的变化。在研究期间收集每日环境PM_（2.5）质量样本，并在实验室中测量29种化学成分。使用正矩阵分解进行环境PM_（2.5）的来源指定，并使用混合效应模型来估计与特定于来源PM_（2.5）浓度相关的心肺作用。七个PM_（2.5）来源被确定为交通排放（12.0％），燃煤（22.0％），次硫酸盐/硝酸盐（30.2％），冶金排放（0.4％），粉尘/土壤（12.4％），工业（6.9 ％）和次要有机气雾剂（9.9％）。与城市校园相比，郊区校园中的环境PM_（2.5）的贡献主要来自中学硫酸盐/硝酸盐（41.8％对22.9％-26.0％）和冶金排放（0.7％对0.3％）。城市校园的PM_（2.5）在交通排放（13.0％-16.3％vs. 5.1％），燃煤（21.0％-30.7％vs. 10.7％）和二次有机气溶胶（9.7％-12.0％）中的贡献更大与8.7％相比）。确定了PM_（2.5）对炎症生物标志物（次硫酸盐/硝酸盐和粉尘/土壤），血压（煤燃烧和冶金排放）和肺功能（粉尘/土壤和工业）的潜在关键来源。使用另一个来源指定工具Unmix进行的分析得出了类似的来源贡献和相关健康影响模式。总之，北京郊区和城市地区的环境PM_（2.5）具有两种不同的源贡献模式，不同来源的PM_（2.5）可能在PM_（2.5）相关的心肺健康效应的不同方面发挥重要作用。

著录项

  • 来源
    《Environmental Science & Technology》 |2014年第6期|3438-3448|共11页
  • 作者

    Shaowei Wu; Furong Deng; Hongying Wei; Jing Huang; Xin Wang; Yu Hao; Chanjuan Zheng; Yu Qin; Haibo Lv; Masayuki Shima; Xinbiao Guo;

  • 作者单位

    Department of Occupational and Environmental Health Sciences, Peking University School of Public Health, Beijing, China;

    Department of Occupational and Environmental Health Sciences, Peking University School of Public Health, Beijing, China;

    Department of Occupational and Environmental Health Sciences, Peking University School of Public Health, Beijing, China;

    Department of Occupational and Environmental Health Sciences, Peking University School of Public Health, Beijing, China;

    Department of Occupational and Environmental Health Sciences, Peking University School of Public Health, Beijing, China;

    Department of Occupational and Environmental Health Sciences, Peking University School of Public Health, Beijing, China;

    Department of Occupational and Environmental Health Sciences, Peking University School of Public Health, Beijing, China;

    Department of Occupational and Environmental Health Sciences, Peking University School of Public Health, Beijing, China;

    Department of Occupational and Environmental Health Sciences, Peking University School of Public Health, Beijing, China;

    Department of Public Health, Hyogo College of Medicine, Hyogo, Japan;

    Department of Occupational and Environmental Health Sciences, Peking University School of Public Health, Beijing, China;

  • 收录信息 美国《科学引文索引》(SCI);美国《工程索引》(EI);美国《生物学医学文摘》(MEDLINE);美国《化学文摘》(CA);
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  • 正文语种 eng
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