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首页> 外文期刊>Journal of Experimental Botany >Expression of ethylene biosynthetic and receptor genes in rose floral tissues during ethylene-enhanced flower opening
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Expression of ethylene biosynthetic and receptor genes in rose floral tissues during ethylene-enhanced flower opening

机译：乙烯增强花开放过程中玫瑰花组织中乙烯生物合成和受体基因的表达

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摘要

Ethylene production, as well as the expression of ethylene biosynthetic (Rh-ACS1–4 and Rh-ACO1) and receptor (Rh-ETR1–5) genes, was determined in five different floral tissues (sepals, petals, stamens, gynoecia, and receptacles) of cut rose (Rosa hybrida cv. Samantha upon treatment with ethylene or the ethylene inhibitor 1-methylcyclopropene (1-MCP). Ethylene-enhanced ethylene production occurred only in gynoecia, petals, and receptacles, with gynoecia showing the greatest enhancement in the early stage of ethylene treatment. However, 1-MCP did not suppress ethylene production in these three tissues. In sepals, ethylene production was highly decreased by ethylene treatment, and increased dramatically by 1-MCP. Ethylene production in stamens remained unchanged after ethylene or 1-MCP treatment. Induction of certain ethylene biosynthetic genes by ethylene in different floral tissues was positively correlated with the ethylene production, and this induction was also not suppressed by 1-MCP. The expression of Rh-ACS2 and Rh-ACS3 was quickly induced by ethylene in gynoecia, but neither Rh-ACS1 nor Rh-ACS4 was induced by ethylene in any of the five tissues. In addition, Rh-ACO1 was induced by ethylene in all floral tissues except sepals. The induced expression of ethylene receptor genes by ethylene was much faster in gynoecia than in petals, and the expression of Rh-ETR3 was strongly suppressed by 1-MCP in all floral tissues. These results indicate that ethylene biosynthesis in gynoecia is regulated developmentally, rather than autocatalytically. The response of rose flowers to ethylene occurs initially in gynoecia, and ethylene may regulate flower opening mainly through the Rh-ETR3 gene in gynoecia.
机译：在五个不同的花卉组织（花萼，花瓣，雄蕊，雌蕊和雌蕊和雌蕊中）测定了乙烯的产生以及乙烯生物合成（Rh-ACS1-4和Rh-ACO1）和受体（Rh-ETR1-5）基因的表达。玫瑰花（Rosa hybrida cv。Samantha）经乙烯或乙烯抑制剂1-甲基环丙烯（1-MCP）处理后，乙烯增强的乙烯生成仅发生在妇科，花瓣和容器中，妇科表现出最大的增强作用。乙烯处理的早期阶段，但是1-MCP并不能抑制这三个组织中的乙烯产生；在萼片中，乙烯处理大大降低了乙烯的产生，而1-MCP则大大增加了乙烯的产生，雄蕊中乙烯的产生保持不变。或1-MCP处理。乙烯在不同花卉组织中对某些乙烯生物合成基因的诱导与乙烯的产生呈正相关，并且这种诱导也未被1-MC抑制。 P.乙烯在绞股蓝中迅速诱导了Rh-ACS2和Rh-ACS3的表达，但是在五个组织中的任何一个中，Rh-ACS1和Rh-ACS4都没有被乙烯诱导。此外，除萼片外，乙烯在所有花卉组织中诱导了Rh-ACO1。雌蕊中乙烯诱导的乙烯受体基因表达比花瓣快得多，并且1-MCP在所有花卉组织中均强烈抑制Rh-ETR3的表达。这些结果表明，女性生殖器中的乙烯生物合成受到发育的调节，而不是自身催化的调节。月季花对乙烯的反应最初发生在妇科，而乙烯可能主要通过妇科中的Rh-ETR3基因调节花的开放。

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