机译:XBP1通过控制HIF1α途径促进三阴性乳腺癌
Sandra and Edward Meyer Cancer Center of Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA,Department of Medicine, Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA;
Centerfor Systems Biomedicine, Division of Digestive Diseases, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, California 90095, USA,Department of Cancer Immunology and AIDS, Dana-Farber Cancer Institute, Boston, Massachusetts 02115, USA;
Lineberger Comprehensive Cancer Center, Department of Pathology and Laboratory Medicine, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina 27599, USA;
Department of Bioinformatics, School of Life Science and Technology, Tongji University, Shanghai 200092, China,Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Ya'an, Sichuan 625014, China;
Departmentof Pathology, Brigham and Women's Hospital, Boston, Massachusetts 02115, USA;
Centerfor Systems Biomedicine, Division of Digestive Diseases, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, California 90095, USA,Department of Cancer Immunology and AIDS, Dana-Farber Cancer Institute, Boston, Massachusetts 02115, USA;
Department of Medical Oncology, Dana-Farber Cancer Institute and Department of Medicine, Harvard Medical School, Boston, Massachusetts 02115, USA;
Whitehead Institute for Biomedical Research, 9 Cambridge Center, Cambridge, Massachusetts 02142, USA;
Department of Medical Oncology, Dana-Farber Cancer Institute and Department of Medicine, Harvard Medical School, Boston, Massachusetts 02115, USA;
Department of Biostatistics and Computational Biology, Dana-Farber Cancer Institute and Harvard School of Public Health, Boston, Massachusetts 02215, USA;
Department of Nanomedicine, Houston Methodist Research Institute, Houston, Texas 77030, USA;
Department of Nanomedicine, Houston Methodist Research Institute, Houston, Texas 77030, USA,Department of Cell and Developmental Biology,Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA;
Endocrine Unit, Massachusetts General Hospital, Boston, Massachusetts 02114, USA;
Sandra and Edward Meyer Cancer Center of Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA,Department of Medicine, Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA;
Division of Hematology/Oncology, Children's Hospital Boston, Boston, Massachusetts 02115, USA;
Sandra and Edward Meyer Cancer Center of Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA,Department of Medicine, Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA;
Sandra and Edward Meyer Cancer Center of Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA,Department of Medicine, Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA;
Houston Methodist Cancer Center, Houston, Texas 77030, USA;
Department of Medicine, Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA,Department of Nanomedicine, Houston Methodist Research Institute, Houston, Texas 77030, USA;
Department of Pathology and Laboratory Medicine, Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA;
Department of Medical Oncology, Dana-Farber Cancer Institute and Department of Medicine, Harvard Medical School, Boston, Massachusetts 02115, USA;
Department of Medicine, Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA,Houston Methodist Cancer Center, Houston, Texas 77030, USA;
Department of Biostatistics and Computational Biology, Dana-Farber Cancer Institute and Harvard School of Public Health, Boston, Massachusetts 02215, USA;
Sandra and Edward Meyer Cancer Center of Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA,Department of Medicine, Weill Cornell Medical College, 1300 York Avenue, New York, New York 10065, USA;
机译:低氧通过IRE1α-XBP1途径诱导miR-153,以微调乳腺癌血管生成中的HIF1α/ VEGFA轴。
机译:LINK-A lncRNA激活三阴性乳腺癌中的常氧HIF1 alpha信号传导
机译:SPHK2 / S1P通过PAK1 / LIMK1 / COFILIN1信号通路促进三阴性乳腺癌转移
机译:使用U-NET架构的三阴性乳腺癌组织病理学图像中的细胞分割
机译:通过药理学和化学治疗干预措施来改善三阴性乳腺癌治疗的代谢途径
机译:XBP1通过控制HIF1α途径促进三阴性乳腺癌
机译:缺氧诱导miR-153通过IS1α-XBP1途径微调HIF1α/ VEGFA轴在乳腺癌血管生成中