机译:释放微生物群的宿主糖可促进肠道后病原体的抗生素扩展
Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305, USA;
Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305, USA;
Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305, USA;
Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305, USA;
Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305, USA,Department of Gastroenterology and Hepatology, Mayo Clinic, Rochester, Minnesota 55905, USA;
Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305, USA;
Glycobiology Research and Training Center, University of California, San Diego, California 92093, USA;
Glycobiology Research and Training Center, University of California, San Diego, California 92093, USA;
department of Population Health and Reproduction, University of California, Davis, California 95616, USA;
Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305, USA;
Department of Microbiology and Immunology, Stanford University School of Medicine, Stanford, California 94305, USA;
机译:宿主介导的糖氧化促进抗生素后病原体的扩展
机译:苹果对青霉菌扩张(病原体)和指状青霉菌(非寄主病原体)感染的差异转录反应
机译:使用粘蛋白衍生的糖的A51赋予促进肠道殖民化的肠道病原体的健身优势
机译:减少进入制糖厂的原糖的色负荷的努力
机译:检查环境,宿主和病原体在牡蛎疱疹病毒和太平洋牡蛎之间的宿主-病原体关系中的作用。
机译:释放微生物群的宿主糖可促进肠道后病原体的抗生素扩展
机译:侵入性森林病原体的关键宿主还促进了California森林在野火的病原体的生存