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首页> 外文期刊>Plant Growth Regulation >Phosphatidylinositol 3-phosphate is required for abscisic acid-induced hydrogen peroxide production in rice leaves
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Phosphatidylinositol 3-phosphate is required for abscisic acid-induced hydrogen peroxide production in rice leaves

机译：脱落酸诱导水稻叶片产生过氧化氢需要磷酸3-磷酸肌醇

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摘要

Rice leaves produce H2O2 in response to abscisic acid (ABA), which results in induction of senescence and accumulation of NH4 +. The upstream steps of the ABA-induced H2O2 production pathway in rice leaves remain largely unclear. In animal cells, H2O2 production in neutrophils is activated by phosphatidylinositol 3-phosphate (PI3P), a product of phosphatidylinositol 3-knase (PI3K). In the present study, we examined whether PI3P plays a role in H2O2 production in rice leaves exposed to ABA. We found that PI3K inhibitors LY 294002 (LY) or wortmannin (WM) inhibited ABA-induced H2O2 production, senescence and NH4 + accumulation. Hydrogen peroxide almost completely rescued the inhibitory effect of LY or WM. It appears that PI3P plays a role in ABA-induced H2O2 production, senescence, and NH4 + accumulation in rice leaves.
机译：水稻叶片响应脱落酸（ABA）而产生H2 O2 ，导致衰老和NH4 + 的积累。 ABA诱导的水稻叶片H2 O2 产生途径的上游步骤仍不清楚。在动物细胞中，嗜中性粒细胞中的H2 O2 产生被磷脂酰肌醇3-激酶（PI3K）的产物磷脂酰肌醇3-磷酸（PI3P）激活。在本研究中，我们研究了PI3P是否在暴露于ABA的水稻叶片中的H2 O2 产生中起作用。我们发现PI3K抑制剂LY 294002（LY）或渥曼青霉素（WM）抑制ABA诱导的H2 O2 产生，衰老和NH4 + 积累。过氧化氢几乎完全拯救了LY或WM的抑制作用。 PI3P似乎在ABA诱导的水稻叶片H2 O2 产生，衰老和NH4 + 积累中起作用。

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