Long-term potentiation increases tyrosine phosphorylation of the N-methyl-D-aspartate receptor subunit 2B in rat dentate gyrus in vivo

Kobi Rosenblum; Yadin Dudai; Gal Richter-Levin

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Long-term potentiation increases tyrosine phosphorylation of the N-methyl-D-aspartate receptor subunit 2B in rat dentate gyrus in vivo

机译：长期增强增强大鼠齿状回体内N-甲基-D-天冬氨酸受体亚基2B的酪氨酸磷酸化

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Long-term potentiation (LTP) is a form of synaptic memory that may subserve developmental and be- havioral plasticity. An intensively investigated form of LTP is dependent upon N-methyl-D-aspartate (NMDA) receptors and can be elicited in the dentate gyrus and hippocampal CA1. Induction of this type of LTP is triggered by influx of Ca~2+ through activated NMDA receptors, but the downstream mechanisms of induction, and even more so of LTP mainte- nance, remain controversial.

机译：长期增强（LTP）是一种突触记忆的形式，可能有助于发育和行为的可塑性。深入研究的LTP形式取决于N-甲基-D-天冬氨酸（NMDA）受体，并且可以在齿状回和海马CA1中引起。这种LTP的诱导是由活化的NMDA受体通过Ca〜2 +的流入而触发的，但是下游的诱导机制，甚至更多的LTP维持机制，仍然存在争议。

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《Proceedings of the National Academy of Sciences of the United States of America》 |1996年第19期|p.10457-10460|共4页
作者
Kobi Rosenblum; Yadin Dudai; Gal Richter-Levin;
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收录信息美国《科学引文索引》(SCI);美国《生物学医学文摘》(MEDLINE);美国《化学文摘》(CA);
原文格式 PDF
正文语种 eng
中图分类自然科学总论;
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专利

1. Enhanced tyrosine phosphorylation of the 2B subunit of the N-methyl-D-aspartate receptor in long-term potentiation [J] . J. A. P. Rostas, V. A. Brent, K. Voss Proceedings of the National Academy of Sciences of the United States of America . 1996,第19期

机译：N-甲基-D-天门冬氨酸受体的2B亚基在长期增强中的酪氨酸磷酸化增强
2. Amygdala N-methyl-D-aspartate receptors participate in the induction of long-term potentiation in the dentate gyrus in vivo. [J] . Ikegaya Y, Saito H, Abe K Neuroscience Letters: An International Multidisciplinary Journal Devoted to the Rapid Publication of Basic Research in the Brain Sciences . 1995,第3期

机译：杏仁核N-甲基-D-天冬氨酸受体参与体内齿状回的长期增强诱导。
3. Deficits in nerve growth factor release and tyrosine receptor kinase phosphorylation are associated with age-related impairment in long-term potentiation in the dentate gyrus. [J] . Kelly A, Maguire C, Lynch MA Neuroscience: An International Journal under the Editorial Direction of IBRO . 2000,第2期

机译：神经生长因子释放和酪氨酸受体激酶磷酸化的缺陷与齿状回的长期增强中与年龄相关的损伤有关。
4. The selective antagonism of A2A adenosine receptors prevents synaptic failure induced by oxygen and glucose deprivation in rat dentate gyrus [C] . Maraulaa G., Trainia C., Mellob T., European Congress of Pharmacology . 2012

机译：A2A腺苷受体的选择性拮抗剂可防止氧气牙齿诱导的氧气失败和葡萄糖剥夺的突触失败
5. Norepinephrine-induced potentiation is not blocked by the local diffusion of the NMDA channel blocker, ketamine, at concentrations that prevent tetanically-induced long-term potentiation in the dentate gyrus in vivo. [D] . Frizzell, Lynn Marie. 1994

机译：去甲肾上腺素诱导的增强作用不会被NMDA通道阻滞剂氯胺酮的局部扩散所阻断，其浓度可防止在体内齿状回中由强直性诱导的长期增强作用。
6. Long-term potentiation increases tyrosine phosphorylation of the N-methyl-D-aspartate receptor subunit 2B in rat dentate gyrus in vivo. [O] . K Rosenblum, Y Dudai, G Richter-Levin 1996

机译：长期增强可增强大鼠齿状回体内N-甲基-D-天冬氨酸受体亚基2B的酪氨酸磷酸化。
7. Long-term potentiation increases tyrosine phosphorylation of the N-methyl-D-aspartate receptor subunit 2B in rat dentate gyrus in vivo. [O] . Rosenblum, K, Dudai, Y, Richter-Levin, G 1996

机译：长期增强可增强大鼠齿状回体内N-甲基-D-天冬氨酸受体亚基2B的酪氨酸磷酸化。
8. Modulation of Long-Term Potentiation and Epileptiform Activity in the Rat Dentate Gyrus by the Group II Metabotropic Glutamate Receptor Subtype mGluR3 [R] . Lea, P. M. 2000

机译：II组代谢型谷氨酸受体亚型mGluR3对大鼠齿状回长期增强和癫痫样活动的调节

Long-term potentiation increases tyrosine phosphorylation of the N-methyl-D-aspartate receptor subunit 2B in rat dentate gyrus in vivo

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