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机译:B-1a中的抗原特异性记忆及其与自然免疫的关系
Department of Genetics, Stanford University School of Medicine, Stanford, CA 94305;
Department of Genetics, Stanford University School of Medicine, Stanford, CA 94305;
Department of Microbiology and Immunology, University of Maryland, Baltimore, MD 21201;
Department of Genetics, Stanford University School of Medicine, Stanford, CA 94305;
Department of Genetics, Stanford University School of Medicine, Stanford, CA 94305;
Department of Microbiology and Immunology, University of Maryland, Baltimore, MD 21201;
Department of Genetics, Stanford University School of Medicine, Stanford, CA 94305;
Department of Genetics, Stanford University School of Medicine, Stanford, CA 94305;
B-1; isotype-switching; memory B cells; inflammation;
机译:B-1a中抗原特异性抗体应答及其与自然免疫的关系
机译:G5PR的转基因过表达通常在中心细胞中增强,从而损害高亲和力抗原特异性B细胞的富集,增加腹膜B-1a细胞并诱导老年雌性小鼠自身免疫
机译:G5PR的转基因过度表达,通常在中心细胞中增强会损害高亲和力抗原特异性B细胞的富集,增加腹膜B-1a细胞,并诱导老年雌性小鼠自身免疫。
机译:单事件不安定性与最近记忆的制造过程之间的关系
机译:记忆CD8 + T细胞在持续的持续感染中仍保持功能性:对抗原特异性免疫重建和增强的影响。
机译:B-1a中的抗原特异性记忆及其与自然免疫的关系
机译:由于自然杀伤细胞的耗尽而导致树突状细胞功能受损,从而破坏了小鼠的抗原特异性免疫反应:抗原脉冲树突状细胞恢复了自然杀伤力较弱的小鼠的适应性免疫