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机译:安非他明致敏需要多巴胺D1受体(D1R)和D2R表达的中棘神经元中平衡的NMDA受体活性
Department of Genome Sciences, University of Washington, Seattle, WA 98195;
Department of Psychiatry and Behavioral Sciences, University of Washington, Seattle, WA 98195;
Howard Hughes Medical Institute, University of Washington, Seattle, WA 98195 Department of Biochemistry, University of Washington, Seattle, WA 98195;
Department of Pharmacology, University of Washington, Seattle, WA 98195;
Departments of Neurology, Pediatrics, and Psychology, and Center on Human Development and Disability, University of Washington,Seattle Children's Hospital, Seattle, WA 98105;
Department of Psychiatry and Behavioral Sciences, University of Washington, Seattle, WA 98195 Howard Hughes Medical Institute, University of Washington, Seattle, WA 98195 Department of Pharmacology, University of Washington, Seattle, WA 98195;
Department of Genome Sciences, University of Washington, Seattle, WA 98195 Howard Hughes Medical Institute, University of Washington, Seattle, WA 98195 Department of Biochemistry, University of Washington, Seattle, WA 98195;
机译:协同相互作用的多巴胺D1和NMDA受体介导大鼠纹状体中棘神经元释放非囊泡转运蛋白依赖性GABA。
机译:多巴胺通过D1和D2多巴胺受体促进纹状体中层棘状神经元的树突状脊柱形成
机译:IP3在体内Amphetamine经验中的受体敏化增强了腹侧特子区域的多巴胺神经元中的NMDA受体可塑性。
机译:设置为纹状体背外侧和腹内侧中棘神经元时,NMDA受体模型在短期和长期内的时间动态影响
机译:第1部分。对非NMDA谷氨酸受体具有活性的分子的设计,合成和结构活性研究:羟苯丙氨酸,喹喔啉二酮和相关分子。第2部分。关于多巴胺氨基和氨基取代与羧酸根阴离子相互作用的计算研究,作为受体相互作用的模型。
机译:安非他明致敏需要多巴胺D1受体(D1R)和D2R表达的中棘神经元中平衡的NMDA受体活性
机译:安非他明致敏需要多巴胺D1受体(D1R)和D2R表达的中棘神经元中平衡的NMDA受体活性