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机译:齿状体育馆中的补偿性网络变化可消除成年小鼠神经发生消融后的长期增效作用
Department of Neurology, University of Michigan Medical School, Ann Arbor, Ml 48109,Neuroscience Graduate Program, University of Michigan Medical School, Ann Arbor, Ml 48109;
Molecular and Behavioral Neuroscience Institute, University of Michigan Medical School, Ann Arbor, Ml 48109;
Department of Neurology, University of Michigan Medical School, Ann Arbor, Ml 48109;
Neuroscience Graduate Program, University of Michigan Medical School, Ann Arbor, Ml 48109,Molecular and Behavioral Neuroscience Institute, University of Michigan Medical School, Ann Arbor, Ml 48109;
Department of Neurology, University of Michigan Medical School, Ann Arbor, Ml 48109,Neuroscience Graduate Program, University of Michigan Medical School, Ann Arbor, Ml 48109,VA Ann Arbor Healthcare System, Ann Arbor, Ml 48105;
Neuroscience Graduate Program, University of Michigan Medical School, Ann Arbor, Ml 48109,Molecular and Behavioral Neuroscience Institute, University of Michigan Medical School, Ann Arbor, Ml 48109,Department of Molecular andIntegrative Physiology, University of Michigan Medical School, Ann Arbor, Ml 48109;
adult neurogenesis; thymidine kinase; metaplasticity; miniature inhibitory postsynaptic currents;
机译:通过体内高频刺激将核糖体蛋白S6定向到树突棘,以诱导齿状回的长期增强。通过体内高频刺激将核糖体蛋白S6定向到树突棘,诱导在齿状回中的长期增强。核糖体蛋白S6通过体内高频刺激诱导齿状回长期增强
机译:缺乏JWA会增强海马齿状回的神经发生和长期增强,从而导致空间认知增强。
机译:缺乏脊柱装置和胸骨细胞器的突触足蛋白缺乏小鼠的齿状回体内的theta-burst长期增强和网络兴奋性受损。
机译:腺苷A / sub 1 / R受体缺乏会增强自由移动小鼠的海马长时程增强
机译:去甲肾上腺素诱导的增强作用不会被NMDA通道阻滞剂氯胺酮的局部扩散所阻断,其浓度可防止在体内齿状回中由强直性诱导的长期增强作用。
机译:幼年成年小鼠神经发生消融后齿状回中的补偿性网络变化恢复了长期增强
机译:幼年成年小鼠神经发生消融后齿状回中的补偿性网络变化恢复了长期增强
机译:II组代谢型谷氨酸受体亚型mGluR3对大鼠齿状回长期增强和癫痫样活动的调节