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机译:异位Myc目标基因JAG2的诱导增强人B细胞模型中的低氧生长和肿瘤发生
Departments of Pediatrics, Johns Hopkins University School of Medicine,Baltimore, MD 21205;
Departments of Pathology,Johns Hopkins University School of Medicine,Baltimore, MD 21205;
Departments of Medicine,Johns Hopkins University School of Medicine,Baltimore, MD 21205;
Departments of Microarray Facility, Johns Hopkins University School of Medicine,Baltimore, MD 21205;
Departments of Medicine,Johns Hopkins University School of Medicine,Baltimore, MD 21205;
Departments of Pathology,Johns Hopkins University School of Medicine,Baltimore, MD 21205;
Comprehensive Cancer Center, Mayo Clinic, Scottsdale, AZ 85259 Department of Medicine, Yong Loo Lin School of Medicine,National University of Singapore, Singapore 545523;
Departments of Pathology,Johns Hopkins University School of Medicine,Baltimore, MD 21205;
Comprehensive Cancer Center, Mayo Clinic, Scottsdale, AZ 85259;
Departments of Pathology,Johns Hopkins University School of Medicine,Baltimore, MD 21205 Departments of Medicine,Johns Hopkins University School of Medicine,Baltimore, MD 21205 Sidney Kimmel Cancer Center, Johns Hopkins University School of Medicine,Baltimore, MD 21205;
neoplasia; notch; target genes; transcription; hypoxia;
机译:在长期培养过程中,在缺乏T细胞的子宫内膜异位症小鼠模型中,其他B细胞缺乏症不会影响异位人类子宫内膜的生长和血管生成
机译:在鼠B细胞淋巴瘤移植模型中反义c-myc和免疫刺激性寡核苷酸抑制肿瘤发生。
机译:回复:Nkx3.1和myc在小鼠和人类前列腺癌发生过程中交叉调节共有的靶基因
机译:血管内皮生长因子的缺氧诱导在突变人SOD1基因的小鼠的脊髓中受损
机译:JP1是一种新的c -Myc靶基因,参与锚定非依赖性生长。
机译:异位Myc目标基因JAG2的诱导增强人B细胞模型中的低氧生长和肿瘤发生
机译:异位Myc目标基因JAG2的诱导增强人B细胞模型中的低氧生长和肿瘤发生