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机译:抗体介导的IL-15阻断可逆转肠上皮细胞中过表达IL-15的转基因小鼠的自身免疫性肠损伤
Department of Allergy and Immunology, Tokyo Metropolitan Institute of Medical Science, Tokyo, 113-8613, Japan;
Department of Allergy and Immunology, Tokyo Metropolitan Institute of Medical Science, Tokyo, 113-8613, Japan;
Metabolism Branch, National Cancer Institute, Bethesda, MD 20892-1374;
Veterans Affairs Medical Center, Washington, DC 20422;
Veterans Affairs Medical Center, Washington, DC 20422;
Department of Pharmacy, Hyogo University of Health Sciences, Kobe, 650-8530, Japan;
Department of Microbiology and Immunology, Osaka University Graduate School of Medicine, Suita, 565-0871, Japan;
Metabolism Branch, National Cancer Institute, Bethesda, MD 20892-1374;
Department of Allergy and Immunology, Tokyo Metropolitan Institute of Medical Science, Tokyo, 113-8613, Japan;
Metabolism Branch, National Cancer Institute, Bethesda, MD 20892-1374;
autoimmunity; celiac disease; immunotherapy; interleukine 15 receptor;
机译:在肠细胞中过表达人IL-15的转基因小鼠概括了B和T细胞介导的乳糜泻的病理表现。
机译:在肠细胞中过表达人IL-15的转基因小鼠概括了乳糜泻的B和T细胞介导的病理表现
机译:可溶性IL-15受体#alpha#-链对IL-15的选择性阻滞增强了心脏异体移植的存活率
机译:小鼠肠道细菌调节衍生自无菌TCR-转基因小鼠的肠免疫细胞的抗原特异性细胞因子产生
机译:经过工程改造以过度表达IL-2,IL-7,IL-15或IL-21的肿瘤特异性人类原代T细胞可在免疫缺陷小鼠模型中赋予不同程度的肿瘤排斥反应和异种移植物抗宿主病。
机译:抗体介导的IL-15阻断可逆转在肠细胞中过表达IL-15的转基因小鼠的自身免疫性肠损伤
机译:抗体介导的IL-15阻断可逆转在肠细胞中过表达IL-15的转基因小鼠的自身免疫性肠损伤