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机译:IκBα促进NF-κB-DNA解离的动力学
Department of Chemistry and Biochemistry, University of California at San Diego, La Jolla, CA 92093-0378;
Department of Chemistry and Biochemistry, University of California at San Diego, La Jolla, CA 92093-0378;
Department of Chemistry and Biochemistry, University of California at San Diego, La Jolla, CA 92093-0378;
Department of Chemistry and Biochemistry, University of California at San Diego, La Jolla, CA 92093-0378;
Department of Chemistry and Biochemistry, University of California at San Diego, La Jolla, CA 92093-0378;
Department of Chemistry and Biochemistry, University of California at San Diego, La Jolla, CA 92093-0378;
binding kinetics; disordered proteins; protein-DNA interaction; transcription; surface plasmon resonance;
机译:GITR的细胞内部分通过Erk和NF-κB信号的反向调节增强NGF促进的神经突生长。GITR的细胞内部分通过Erk和NF-κB信号的反向调节增强NGF促进的神经突生长。 GITR的细胞内部分通过Erk和NF-κB信号转导的反向调节增强NGF促进的神经突生长。
机译:辛伐他汀对兔动脉粥样硬化模型中NF-κB-DNA结合活性和单核细胞趋化蛋白-1表达的影响
机译:在半稀释制度中增强遥甲基吡啶封端的聚(乙二醇)组件中的金属 - 超分子解离动力学
机译:用尿红酸抑制NF-κB的增强紫杉醇诱导的细胞凋亡
机译:通过使用微流控芯片增强了Jurkat T细胞中NF-κB信号转导通路和荧光素形成/流出的生化动力学研究
机译:IκBα促进NF-κB·DNA解离的动力学
机译:IκBα促进NF-κB·DNA解离的动力学
机译:基于共振增强多光子电离的极低压光解条件下原位自由基检测。六氟丙酮红外多光子解离产生CF3自由基的动力学