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首页> 外文期刊>Proceedings of the National Academy of Sciences of the United States of America >RID1, encoding a Cys2/His2-type zinc finger transcription factor, acts as a master switch from vegetative to floral development in rice
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RID1, encoding a Cys2/His2-type zinc finger transcription factor, acts as a master switch from vegetative to floral development in rice

机译：RID1编码Cys2 / His2型锌指转录因子，可作为水稻从营养发育到花卉发育的主要开关

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摘要

Transition from the vegetative phase to reproductive phase is a crucial process in the life cycle of higher plants. Although the molecular mechanisms of flowering regulation have been extensively charac-terized in a number of plant species, little is known regarding how the transition process initiates. Here, we show that the Rice Indeterminate 1 (RID1) gene acts as the master switch for the transition from the vegetative to reproductive phase. RID1 encodes a Cys-2/His-2-type zinc finger transcription factor that does not have an ortholog in Arabidopsis spp. A RID1 knockout (rid1), mutated by T-DNA insertion, never headed after growing for > 500 days under a range of growth conditions and is thus referred to as a never-flowering phenotype. This mutation-suppressed expression of the genes is known to be involved in flowering regulation, especially in the Ehd1/Hd3a pathway and a series of RFT homologs. RID1 seems to be independent of the circadian clock. A model was proposed to place RID1 in the molecular pathways of flowering regulation in rice, for which there are two indispensable elements. In the first, RID1 is controlling the phase transition and initiation of floral induction. In the other, the Hd3a/RFL1/FTL complex acts as the immediate inducer of flowering. Loss of function in either element would cause never-flowering. Once the phase transition is induced with the activation of RID1, flowering signal is transduced and regulated through the various pathways and eventually integrated with FT-like proteins to induce flowering.
机译：从营养阶段到生殖阶段的转变是高等植物生命周期中的关键过程。尽管开花调节的分子机制已在许多植物物种中得到广泛表征，但对于过渡过程的启动方式知之甚少。在这里，我们显示水稻不确定1（RID1）基因充当从营养期到生殖期过渡的主要开关。 RID1编码在拟南芥属物种中没有直系同源物的Cys-2 / His-2-型锌指转录因子。通过T-DNA插入突变的RID1基因敲除（rid1），在一系列生长条件下生长> 500天后再也没有出现过，因此被称为永不开花的表型。已知这种基因的突变抑制表达与开花调控有关，特别是在Ehd1 / Hd3a途径和一系列RFT同源物中。 RID1似乎与生物钟无关。提出了一个将RID1置于水稻开花调控分子途径的模型，其中有两个必不可少的要素。首先，RID1控制相变和花诱导的启动。另一方面，Hd3a / RFL1 / FTL复合体充当开花的直接诱导剂。任一元素的功能丧失都会导致永不开花。一旦通过RID1的激活诱导了相变，就通过各种途径转导并调节了开花信号，并最终与FT-like蛋白整合以诱导开花。

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