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首页> 外文期刊>Soil Science and Plant Nutrition >O2-enhanced induction of photosynthesis in rice leaves: the Mehler-ascorbate peroxidase (MAP) pathway drives cyclic electron flow within PSII and cyclic electron flow around PSI
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O2-enhanced induction of photosynthesis in rice leaves: the Mehler-ascorbate peroxidase (MAP) pathway drives cyclic electron flow within PSII and cyclic electron flow around PSI

机译：O 2 增强水稻叶片光合作用的诱导：梅勒-抗坏血酸过氧化物酶（MAP）途径驱动PSII中的循环电子流和PSI周围的循环电子流

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Lowering the oxygen (O2) partial pressure from 21a??kPa to 1a??kPa delayed the light-dependent increase of the net carbon dioxide (CO2) assimilation rate in rice (Oryza sativa L. cv. Notohikari) leaves. Researching the underlying molecular mechanisms that act before the start of photosynthesis, we established the following facts. First, O2 at 21a??kPa enhanced the quantum yield of PSII [Y(II)] and PSI [Y(I)]. More than 90% of Y(II) and Y(I) were not accounted for by O2-dependent electron flow in the Mehler-ascorbate peroxidase (MAP) pathway. Both yields increased further with the start of photosynthesis. Second, O2 enhanced photochemical quenching of chlorophyll (Chl) fluorescence (qL). qL also increased further with the rate of photosynthesis. Third, O2 enhanced the photo-oxidation of P700. Fourth, O2 suppressed the reduction of P700. Fifth, O2 enhanced non-photochemical quenching of Chl fluorescence (NPQ). These results showed that the MAP pathway triggered cyclic electron flow within PSII (CEF-II) and cyclic electron flow around PSI (CEF-I) by inducing ??pH across thylakoid membranes and oxidizing the plastoquinone pool, before photosynthesis started. We propose that the photosynthetic electron transport system is controlled by the MAP pathway, which would explain the O2-dependent enhancement of the induction of photosynthesis.View full textDownload full textKeywordscyclic electron flow (CEF), Mehler-ascorbate peroxidase (MAP) pathway, O2 , rice, rubisco.Related var addthis_config = { ui_cobrand: "Taylor & Francis Online", services_compact: "citeulike,netvibes,twitter,technorati,delicious,linkedin,facebook,stumbleupon,digg,google,more", pubid: "ra-4dff56cd6bb1830b" }; Add to shortlist Link Permalink http://dx.doi.org/10.1080/00380768.2012.736078
机译：将氧气（O 2 ）分压从21a ?? kPa降低到1a ?? kPa延迟了净二氧化碳（CO 2 ）同化率的光依赖性增加在水稻（Oryza sativa L. cv。Notohikari）叶中。研究光合作用开始之前起作用的潜在分子机制，我们建立了以下事实。首先，在21a ?? kPa处的O 2 增强了PSII [Y（II）]和PSI [Y（I）]的量子产率。在Mehler-抗坏血酸过氧化物酶（MAP）途径中，O 2 依赖的电子流不能解释超过90％的Y（II）和Y（I）。随着光合作用的开始，两种产量都进一步增加。其次，O 2 增强了叶绿素（Chl）荧光（qL）的光化学猝灭。 qL也随着光合作用的速率进一步增加。第三，O 2 增强了P700的光氧化作用。第四，O 2 抑制了P700的还原。第五，O 2 增强了Chl荧光（NPQ）的非光化学猝灭。这些结果表明，在光合作用开始之前，MAP途径通过在类囊体膜上诱导ΔεpH并氧化质体醌池，从而触发了PSII内的循环电子流（CEF-II）和PSI周围的循环电子流（CEF-I）。我们认为光合作用电子传输系统受MAP路径控制，这可以解释O 2 依赖于光合作用的增强作用。查看全文下载全文关键词循环电子流（CEF），Mehler-抗坏血酸过氧化物酶（MAP）途径，O2，大米，rubisco.related var addthis_config = {ui_cobrand：“泰勒和弗朗西斯在线”，servicescompact：“ citeulike，netvibes，twitter，technorati，delicious，linkedin，facebook，stumbleupon，digg，google，更多”，发布号：“ ra-4dff56cd6bb1830b”};添加到候选列表链接永久链接http://dx.doi.org/10.1080/00380768.2012.736078

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