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首页> 外文期刊>Journal of Virology >Infection by Bacteriophage P1 and Development of Host-controlled Restriction and Modification and of Lysogenic Immunity
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Infection by Bacteriophage P1 and Development of Host-controlled Restriction and Modification and of Lysogenic Immunity

机译：通过噬菌体P1感染以及宿主控制的限制和改性和溶血性免疫的发展

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Shigella dysenteriae cells were infected with phage P1 or P1cl. The outcome of superinfection of these cells with phage T1.Sh or T1.Sh(P1) or P1cl was studied as a function of time after the initial infection. Cells undergoing either a lytic response or a lysogenic response to the primary infection develop the ability to specifically restrict T1.Sh between 30 and 45 min. Between 15 and 30 min, the cells seem to develop the ability to produce T1.Sh(P1) after infection by T1.Sh. However, reasons are given for believing that this apparent time difference is consistent with a simultaneous development of the two capacities (restriction and modification) within the cell. This development occurs between 30 and 45 min. Cells infected with P1cl and superinfected 45 or more min later with T1.Sh(P1) can yield both P1cl and T1. Cells infected with P1 become resistant to infection by P1cl within 5 to 10 min. It is argued that this early immunity is not necessarily different in mechanism from true lysogenic immunity.
机译： shigella dysenteriae 细胞用噬菌体p1或p1cl感染。用噬菌体T1.SH或T1.SH（P1）或P1CL的这些细胞的超细化的结果作为初始感染后的时间。经历裂解反应或对原发性感染的溶血性反应的细胞会产生特异性限制30至45分钟的能力。在15至30分钟之间，细胞似乎在通过T1.Sh感染后产生产生T1.Sh（P1）的能力。然而，为了相信这种表观时间差的原因是与细胞内的两个能力（限制和修饰）同时发展一致。这种发展发生在30到45分钟之间。用T1.SH（P1）以后用P1Cl和SuperInfected 45或更多Min感染的细胞可以产生P1Cl和T1。用P1感染的细胞对5至10分钟内P1Cl感染变得抗性。认为，这种早期免疫不一定在真正溶血性免疫的机制中不同。

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