Endoplasmic reticulum stress response as a possible mechanism of cyclooxygenase-2-independent anticancer effect of celecoxib.

Wonjae Cha; Seok-Woo Park; Tack-Kyun Kwon; J Hun Hah; Myung-Whun Sung

首页> 外文期刊>Anticancer Research: International Journal of Cancer Research and Treatment >Endoplasmic reticulum stress response as a possible mechanism of cyclooxygenase-2-independent anticancer effect of celecoxib.

【24h】

Endoplasmic reticulum stress response as a possible mechanism of cyclooxygenase-2-independent anticancer effect of celecoxib.

机译：内质网应激反应是塞来昔布不依赖环氧合酶2的抗癌作用的可能机制。

获取原文

获取原文并翻译 | 示例

获取外文期刊封面封底 >>

开具论文收录证明 >>

文献代查 >>

页面导航

摘要
著录项
相似文献
相关主题

摘要

ER stress response could be a COX2-independent anticancer mechanism of celecoxib.

机译：ER应激反应可能是塞来昔布不依赖COX2的抗癌机制。

著录项

来源
《Anticancer Research: International Journal of Cancer Research and Treatment》 |2014年第4期|共5页
作者
Wonjae Cha; Seok-Woo Park; Tack-Kyun Kwon; J Hun Hah; Myung-Whun Sung;
展开▼
作者单位

展开▼
收录信息
原文格式 PDF
正文语种 eng
中图分类
关键词

相似文献

外文文献
中文文献
专利

1. Endoplasmic reticulum stress response as a possible mechanism of cyclooxygenase-2-independent anticancer effect of celecoxib. [J] . Wonjae Cha, Seok-Woo Park, Tack-Kyun Kwon, Anticancer Research: International Journal of Cancer Research and Treatment . 2014,第4期

机译：内质网应激反应作为Celecoxib的环氧氧基酶-2-2无抗癌作用的可能机制。
2. Endoplasmic reticulum stressed by pollution. Focus on "Airborne particulate matter selectively activates endoplasmic reticulum stress response in the lung and liver tissues". [J] . Velasco G American Journal of Physiology . 2010,第4aPta1期

机译：内质网受污染。专注于“空气中的颗粒物选择性激活肺和肝组织中的内质网应激反应”。
3. Calcium-activated endoplasmic reticulum stress as a major component of tumor cell death induced by 2,5-dimethyl-celecoxib, a non-coxib analogue of celecoxib. [J] . Pyrko P, Kardosh A, Liu YT, Molecular cancer therapeutics . 2007,第4期

机译：钙激活的内质网应激是2,5-二甲基-celecoxib（一种celecoxib的非coxib类似物）诱导的肿瘤细胞死亡的主要成分。
4. Effects on endoplasmic reticulum stress response of applying nanosecond pulsed electric fields [C] . Fukuda Hiroshi, Miyake Masato, Hirai Hiroto, IEEE Pulsed Power Conference . 2015

机译：纳秒脉冲电场的内质网应力响应的影响
5. The mechanisms and function of regulated Ire1-dependent decay during endoplasmic reticulum stress. [D] . Moore, Kristin A. 2016

机译：内质网应激过程中调节的Ire1依赖性衰变的机制和功能。
6. A Mouse Model Suggests Two Mechanisms for Thyroid Alterations in Infantile Cystinosis: Decreased Thyroglobulin Synthesis Due to Endoplasmic Reticulum Stress/Unfolded Protein Response and Impaired Lysosomal Processing [O] . H. P. Gaide Chevronnay, V. Janssens, P. Van Der Smissen, -1

机译：小鼠模型提示小儿膀胱病变中甲状腺改变的两种机制：由于内质网应激/未折叠的蛋白质反应和溶酶体加工受损甲状腺球蛋白合成减少
7. A mouse model suggests two mechanisms for thyroid alterations in infantile cystinosis: decreased thyroglobulin synthesis due to endoplasmic reticulum stress/unfolded protein response and impaired lysosomal processing. [O] . Gaide Chevronnay, Héloïse P., Janssens, V, Van Der Smissen, Patrick, 2015

机译：小鼠模型提示婴儿型胱氨酸病中甲状腺改变的两种机制：由于内质网应激/未折叠的蛋白质反应和溶酶体加工受损，甲状腺球蛋白合成减少。

Endoplasmic reticulum stress response as a possible mechanism of cyclooxygenase-2-independent anticancer effect of celecoxib.

摘要

著录项

相似文献

相关主题

期刊订阅