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Editorial comment

机译：编辑评论

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In this volume on neuroanesthesia and neurocritical care, we have assembled six articles on seemingly unconnected topics. However, despite the appearance, I believe they illustrate the broad spectrum of the knowledge base and the scope of practice of the neuroanesthe-siologisteurointensivist. The knowledge base spans from basic science with understanding of the anatomical basis of loss of consciousness with induction of anesthesia as demonstrated by functional imaging, as well as the effects of anesthetic agents on cerebral blood flow and metabolism (Drs Ramani and Wardhan; pp. 530-536) to clinical techniques and neuromonitoring. An increasingly sophisticated imaging paradigm has allowed us to peer into the site of action of anesthetic agents in a dynamic fashion that heretofore could not even be imagined, let alone studied. Although not addressed in this volume, other molecular imaging methods may some day help us to understand the actions of propofol on mitochondria that place patients at risk of arare but potentially fatal reaction to a widely used anesthetic, propofol (paper by Drs Cremer and Kalkman; pp.544-551).
机译：在这本关于神经麻醉和神经重症监护的文章中，我们整理了六篇关于看似无关的主题的文章。但是，尽管出现了，但我相信它们说明了广泛的知识基础以及神经麻醉专家/神经强化专家的实践范围。该知识库涵盖了基础科学领域，了解功能成像显示的麻醉诱导导致的意识丧失的解剖学基础，以及麻醉剂对脑血流和新陈代谢的影响（Ramani和Wardhan博士;第530页） -536）用于临床技术和神经监测。日益复杂的成像范例使我们能够以动态方式凝视麻醉剂的作用部位，这是迄今为止甚至无法想象的，更不用说研究了。尽管在本卷中未作介绍，但其他分子成像方法有一天可能会帮助我们了解丙泊酚对线粒体的作用，使患者处于自发危险，但对广泛使用的麻醉剂丙泊酚具有致命的反应（Cresmer和Kalkman博士的论文; 544-551页）。

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