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首页> 外文期刊>Hepatology research: the official journal of the Japan Society of Hepatology >A case of adenomatous hyperplasia in the liver with chronic hepatitis C that transformed into hepatocellular carcinoma.
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A case of adenomatous hyperplasia in the liver with chronic hepatitis C that transformed into hepatocellular carcinoma.

机译：肝腺瘤的增生转化为慢性丙型肝炎肝细胞癌。

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摘要

We report a rare case of adenomatous hyperplasia (AH) with malignant transformation, which was observed initially in the liver with chronic hepatitis C. A 47-year-old man was admitted to the hospital and chronic active hepatitis was diagnosed using fine needle biopsy, in addition abdominal computed tomography (CT) and ultrasonography showed multiple and hypovascular liver mass. Needle biopsy of the nodules showed AH. Despite transcatheter arterial embolization and percutaneous ethanol injection therapy (PEIT), the patient was readmitted due to enlargement of the nodules and elevation of alpha-fetoprotein 9 months later. On readmission, the nodules in the liver showed early-stage well-differentiated hepatocellular carcinoma (HCC) histopathologically, that were strongly stained on CT-arteriography and were stained less than the surrounding liver on CT-arterial portography, indicating arterial supply in the nodules and malignant transformation. Thereafter, the patient developed jaundice, and enhanced CT and magnetic resonance imaging showed multiple nodules, which occupied most of the liver, and needle biopsy revealed typical well-differentiated HCC. The patient died of hepatic failure 3 years after the initial admission. In this case, it was confirmed that AH transformed into HCC, which showed a multiple tumor on pathological diagnosis. It has been reported that borderline lesions are curable by less aggressive procedures such as enucleation and PEIT. However, such procedures may not be useful for this type of HCC, which rapidly developed from borderline lesions of the liver.
机译：我们报告一个罕见的腺瘤的增生(啊)恶性转化,这是观察到最初与慢性肝脏肝炎c . 47岁男子承认医院和慢性活动性肝炎使用细针活检诊断,此外腹部计算机断层扫描(CT)和超声显示多个hypovascular肝脏质量。啊。和经皮酒精注射治疗(PEIT),病人再次入院结节和海拔的扩大甲胎蛋白9个月后。在肝脏结节显示早期分化良好型的肝细胞癌(HCC)组织病理学,强烈彩色CT-arteriography和低染色比周围CT-arterial肝脏门静脉造影术,表明动脉供应结节和恶性转变。患者黄疸,开发和增强CT磁共振成像显示,多个占领了大部分的肝脏结节,和针吸活组织检查显示典型的分化良好型的肝癌。肝衰竭3年在最初入学。显示多个转变为肝癌肿瘤病理诊断。报道称,临界病变是可以治愈的不那么咄咄逼人摘出术等手术和PEIT。对于这种类型的肝癌,迅速由临界病变的肝脏。

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