Hepatitis B virus-cell interactions and pathogenesis.

Nguyen DH; Ludgate L; Hu J

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Hepatitis B virus-cell interactions and pathogenesis.

机译：乙肝virus-cell交互和发病机理。

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摘要

Like all viruses, hepatitis B virus (HBV) replication and pathogenesis depends on the critical interplay between viral and host factors. In this review, we will focus on the recent progress in understanding the virus-host interactions at the level of the infected cell. These interactions include the requirement of cellular chaperones for the initiation of HBV reverse transcription, the role of the HBV X protein (HBx) in modifying viral and cellular transcription and signaling, the formation of the HBV episomal DNA and its epigenetic regulation in viral persistence, and the cellular factors involved in viral entry, nucleocapsid maturation, and virion secretion.

机译：像所有的病毒,乙型肝炎病毒(HBV)复制和发病机制取决于关键病毒和宿主之间的相互作用的因素。最近的进步在理解病毒-宿主交互在感染细胞的水平。这些交互包括的要求细胞起始陪伴的乙肝病毒反转录,乙肝病毒X的角色在修改病毒和细胞蛋白(HBx)转录和信号的形成乙肝病毒游离DNA及其表观遗传调控病毒的持久性和细胞因子参与病毒,核衣壳成熟,和病毒粒子的分泌。

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来源
《Journal of Cellular Physiology》 |2008年第2期|289-294|共6页
作者
Nguyen DH; Ludgate L; Hu J;
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作者单位

Department of Microbiology and Immunology, The Pennsylvania State University, College of Medicine, Hershey, Pennsylvania 17033, USA.;

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原文格式 PDF
正文语种英语
中图分类细胞生物学;
关键词
Cell Communication; Hepatitis B virus; Gene Expression RegulationpathogenesisDNAcircular;

机译：细胞通讯,乙型肝炎病毒;基因表达RegulationpathogenesisDNAcircular;

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Hepatitis B virus-cell interactions and pathogenesis.

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