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首页> 外文期刊>Human psychopharmacology: clinical and experimental >Alcohol continues to affect sleepiness related driving impairment, when breath alcohol levels have fallen to near-zero.
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Alcohol continues to affect sleepiness related driving impairment, when breath alcohol levels have fallen to near-zero.

机译：酒精继续影响睡眠相关开车的障碍,当呼吸酒精含量已降至接近零。

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摘要

Epidemiological findings point to very low blood alcohol levels heightening the risk of sleep-related fatal road crashes. This was further assessed using a full sized interactive car simulator. Twenty, sleep restricted, healthy young men underwent a 2 h simulated afternoon monotonous drive, having previously consumed nil alcohol or 3 units >90 min previously, and having near-zero breath alcohol (BrACs) at the start of the drive. In a repeated measures, double-blind, balanced design, driving performance, subjective sleepiness and EEG were monitored throughout. Compared with nil alcohol, the alcohol condition initially increased sleepiness-related driving impairment. However, this was not mirrored by subjective sleepiness or EEG. An unexpected reversal (i.e. improvement) in driving impairment occurred with the alcohol group, in the second hour of the drive. This was supported by a trend for improved subjective alertness. Alcohol continued to interact with sleepiness-related driving impairment after BrACshad reached zero. However, a lack of subjective perception of increased sleepiness, at this time, further points to the dangerous combination of even modest alcohol intake and sleepiness, and confirms the road crash findings. BrACs are a poor guide to driver impairment. Copyright 2004 John Wiley & Sons, Ltd.
机译：流行病学调查结果指向非常低的血液酒精含量增加的风险睡眠致命的交通事故。使用一个全尺寸的互动进一步评估汽车模拟器。年轻人接受了下午2 h模拟单调的驱动器,在之前消耗零酒精或3单位> 90分钟以前,和拥有接近于零的呼吸酒精(brac)开始的驱动器。平衡设计、驾驶性能、主观的嗜睡和脑电图监测。与零酒精相比,酒精的条件最初增加瞌睡驾驶障碍。主观嗜睡或脑电图。反转(即改进)在推动障碍发生与酒精组,在第二小时的车程。提高主观的警觉性。继续与瞌睡开车后损伤BrACshad达到零。然而,缺乏主观的看法嗜睡,此时,进一步指向的危险组合适度饮酒和嗜睡证实了道路事故的发现。可怜的司机障碍指南。约翰•威利& Sons有限公司

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