WCN23-1224 Blocking Plasminogen Activator Inhibitor 1 (PAI-1) ameliorates TGFβ-induced Phenotype Transition of Peritoneal Mesothelial Cells (MCs)

D. A. Kim; H. J. Kang; M. YuD. H. Kang

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WCN23-1224 Blocking Plasminogen Activator Inhibitor 1 (PAI-1) ameliorates TGFβ-induced Phenotype Transition of Peritoneal Mesothelial Cells (MCs)

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The epithelial-to-mesenchymal transition (EMT) of MCs is an early mechanism of peritoneal dysfunction in peritoneal dialysis (PD). Plasminogen activator inhibitor-1 (PAI-1) is an inhibitor of fibrinolysis by hindering the proteolytic activity of tissue type plasminogen activator, and recently reported to regulate EMT of cancer cells. The nuclear factor of activated T cells (NFAT5) is known to regulate PAI-1 transcription, and also to promote EMT of cancer cells. The aim of this study is to investigate the role of PAI-1 and NFAT5 in TGFβ-induced EMT of MCs and its mechanism.

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《asian journal of surgery》 |2023年第3期|366-367|共2页
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D. A. Kim; H. J. Kang; M. YuD. H. Kang;
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Ewha Womans University School of Medicine,Nephrology;

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WCN23-1224 Blocking Plasminogen Activator Inhibitor 1 (PAI-1) ameliorates TGFβ-induced Phenotype Transition of Peritoneal Mesothelial Cells (MCs)

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