Receptor Changes in Endotoxemia.

机译：受体内毒素血症的变化。

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Endotoxemia and sepsis, whether occurring in an experimental animal or in a patient in a clinical setting are accompanied by significant displacements in the regulation of hemodynamic cardiovascular status, metabolism and immunocompetence. With such widespread oscillations in several homeostatic mechanisms one would expect accompanying changes in the functioning of the appropriate hormones, neurotransmitters and mediators that are responsible for orchestrating the modulation of the internal environment. Various agonists and antagonists have been used for many years in attempts to ameliorate the deleterious effects of endotoxemia and sepsis. Dopamine has been used primarily as an inotropic agent in septic shock. Although the use of corticosteroids in the treatment of septic shock remains controversial, several investigators have found them to be effective. In experimental endotoxic shock TRH has been shown to improve cardiovascular pathophysiology. Reprints. (kt)

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Spitzer, J. A.; Rodriguez De Turco, E. B.; Deaciuc, I. V.; Roth, B. L.; Hermiller, J. B.;
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年度 1989
页码 1-13
总页数 13
原文格式 PDF
正文语种 eng
中图分类工业技术;
关键词
Endotoxemia; Cardiovascular system; Clinical medicine; Corticosteroid agents; Displacement; Dopamine; Endotoxic shock; Environments; Homeostasis; Hormones; Infectious diseases; Internal; Laboratory animals; Metabolism; Microorganisms; Modulation; Neuromuscular transmission; Oscillation; Pathophysiology; Poisoning; Reprints; Sense organs; Shock(Pathology); Neurochemical transmission; Endogenous substances receptors; Sepsis; Neurotransmitters; Mediators; TRH(Thyrotropin Releasing Hormone); Inotropic Agents;

机译：内毒素血症;心血管系统;临床医学;皮质类固醇药物;置换;多巴胺;内毒素休克;环境;体内平衡;激素;传染病;内部;实验室动物;代谢;微生物;调节;神经肌肉传递;振荡;病理生理学;中毒;重印;感觉器官;休克（病理学）;神经化学传递;内源性物质受体;脓毒症;神经递质;介质; TRH（促甲状腺激素释放激素）;正性肌力药;

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专利

1. Expression of human immunodeficiency virus coreceptors CXC chemokine receptor 4 and CC chemokine receptor 5 on monocytes is down-regulated during human endotoxemia. [J] . Juffermans NP, Weijer S, Verbon A, The Journal of Infectious Diseases . 2002,第7期

机译：人类内毒素血症期间，人类免疫缺陷病毒共受体CXC趋化因子受体4和CC趋化因子受体5在单核细胞上的表达下调。
2. P38 mitogen activated protein kinase is involved in the downregulation of granulocyte CXC chemokine receptors 1 and 2 during human endotoxemia. [J] . Van Den Blink B, Branger J, Weijer S, Journal of Clinical Immunology . 2004,第1期

机译：P38促分裂原活化蛋白激酶参与人类内毒素血症期间粒细胞CXC趋化因子受体1和2的下调。
3. Epinephrine attenuates down-regulation of monocyte tumor necrosis factor receptors during human endotoxemia. [J] . van der Poll T, Calvano SE, Kumar A, Journal of Leukocyte Biology: An Official Publication of the Reticuloendothelial Society . 1997,第2期

机译：肾上腺素减弱人内毒素血症期间单核细胞肿瘤坏死因子受体的下调。
4. Expression Study of Estrogen Receptor-related Receptors and Steroid Hormone Receptors in Human Prostatic Cells [C] . Franky L. Chan, C.P. Cheung, Lung-Wai Chan, International Symposium on Hormonal Carcinogenesis . 2005

机译：雌激素受体相关受体和类固醇激素受体在人前列腺细胞中的表达研究
5. Heart rate variability and rhythmic influences in human endotoxemia. [D] . Scheff, Jeremy D. 2013

机译：人类内毒素血症的心率变异性和节律性影响。
6. Interleukin-1 receptor blockade improves survival and hemodynamic performance in Escherichia coli septic shock but fails to alter host responses to sublethal endotoxemia. [O] . E Fischer, M A Marano, K J Van Zee, 1992

机译：白介素-1受体阻滞剂可提高大肠杆菌败血症性休克的存活率和血液动力学性能但不能改变宿主对亚致死性内毒素血症的反应。
7. Bone marrow and nonbone marrow Toll like receptor 4 regulate acute hepatic injury induced by endotoxemia. [O] . Edith Hochhauser, Orna Avlas, Reut Fallach, 2013

机译：骨髓和非骨髓Toll样受体4调节内毒素血症引起的急性肝损伤。

Receptor Changes in Endotoxemia.

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