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首页> 美国政府科技报告 >Investigations in Fish Control: 92. Acute and Chronic Toxicity of Rotenone to 'Daphnia magna'. 93. Toxicity of Rotenone to Developing Rainbow Trout. 94. Oral Toxicity of Rotenone to Mammals
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Investigations in Fish Control: 92. Acute and Chronic Toxicity of Rotenone to 'Daphnia magna'. 93. Toxicity of Rotenone to Developing Rainbow Trout. 94. Oral Toxicity of Rotenone to Mammals

机译：鱼类控制调查：92。鱼藤酮对'大型蚤'的急性和慢性毒性。 93.鱼藤酮对发育虹鳟鱼的毒性。 94.鱼藤酮对哺乳动物的口服毒性

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The continued use of rotenone as a fish toxicant depends on the development of information requested by the U.S. Environmental Protection Agency for reregistration. To meet one of the requirements, the authors exposed Daphia magna to rotenone in toxicity tests. In exposure of 0.5 to 10 micrograms/L rotenone in a 48-h acute toxicity test, the EC50 was 3.7 micrograms/L; in exposures of 0.312 to 5.0 micrograms/L in a 21-day chronic toxicity test, the EC50 was 2.1 micrograms/L. The no-observed-effect concentration was 1.25 micrograms/L. In a flow-through toxicity test, the authors exposed eyed eggs and larvae of rainbow trout (Salmo gairdneri) for 32 days to crystalline technical grade rotenone to determine its effects on survival, hatching, and growth. Rotenone concentrations of 1.0 to 10.0 micrograms/L did not kill the eyed eggs which hatched normally after 5 or 6 days of exposure. Information required by the U.S. Environmental Protection Agency to support the continued use of rotenone as a fish toxicant includes data on toxicity to nontarget organisms. Information summarized here was developed in three mammalian safety studies of rotenone: chronic oral toxicity in rats, effects on reproduction in rats and subchronic oral toxicity in dogs.

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