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Hypoxia Inhibits Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand-Induced Apoptosis by Blocking Bax Translocation

机译：缺氧抑制肿瘤坏死因子相关的凋亡诱导的诱导配体诱导的细胞凋亡通过阻断BAX易位

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Moonil Kim; Sang-Youel Park; Hyun-Sook Pai; Tae-Hyoung Kim; Timothy R. Billiar; Dai-Wu Seol;
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年度 2004
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1. Hypoxia-mediated activation of autophagic flux inhibits apoptosis of keratinocytes via blocking tumor necrosis factor-related apoptosis-inducing ligand [J] . Kim Sung-Wook, Park Sang-Youel Molecular medicine reports . 2016,第1aPta2期

机译：缺氧介导的自噬通量激活通过阻断肿瘤坏死因子相关的凋亡诱导配体抑制角质形成细胞的凋亡
2. Enforced expression of a truncated form of Bax-alpha (tBax) driven by human telomerase reverse transcriptase (hTERT) promoter sensitizes tumor cells to chemotherapeutic agents or tumor necrosis factor-related apoptosis-inducing ligand (TRAIL). [J] . Toyota H, Kondo S, Kyo S, Anticancer Research: International Journal of Cancer Research and Treatment . 2006,第1Appa期

机译：由人类端粒酶逆转录酶（hTERT）启动子驱动的Bax-α（tBax）截短形式的强制表达使肿瘤细胞对化学治疗剂或肿瘤坏死因子相关的凋亡诱导配体（TRAIL）敏感。
3. Telomerase reverse transcriptase interference synergistically promotes tumor necrosis factor-related apoptosis-inducing ligand-induced oral squamous cell carcinoma apoptosis and suppresses proliferation in vitro and in vivo [J] . Zhao Xin, Zhang Cuicui, Le Zhiliang, International journal of molecular medicine . 2018,第3期

机译：端粒酶逆转录酶干扰协同促进肿瘤坏死因子相关的细胞凋亡诱导配体诱导的口腔鳞状细胞癌细胞凋亡并在体外和体内抑制增殖
4. Effect of Molecular Size of PEGylated tumor necrosis factor factor-related apoptosis apoptosis-inducing ligand(TRAIL) : T The Biological Activity and he Stability [C] . Tae Hyung Kim, Su Young Chae, Seung Min Byun, Annual meeting exposition of the Controlled Release Society . 2009

机译：PEG化肿瘤坏死因子相关细胞凋亡诱导配体（TRAIL）分子大小的影响生物学活性和稳定性
5. Impact of Hepatitis C Virus Core Protein on the Tumor Necrosis Factor Apoptosis-inducing Ligand Pathway in Peripheral Blood Mononuclear Cells [D] . Guy, Janelle. 2018

机译：丙型肝炎病毒核心蛋白对外周血单个核细胞中肿瘤坏死因子凋亡诱导配体途径的影响
6. Expression of Concern: Notch1 signaling sensitizes tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis in human hepatocellular carcinoma cells by inhibiting Akt/Hdm2-mediated p53 degradation and up-regulating p53-dependent DR5 expression. [O] . 2020

机译：关注的表达：Notch1信号通过抑制Akt / Hdm2介导的p53降解并上调p53依赖的DR5表达从而使人肝癌细胞中肿瘤坏死因子相关的凋亡诱导配体诱导的细胞凋亡敏感。
7. Expression of Concern: Notch1 signaling sensitizes tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis in human hepatocellular carcinoma cells by inhibiting Akt/Hdm2-mediated p53 degradation and up-regulating p53-dependent DR5 expression. [O] . 2020

机译：令人担忧的表达：Notch1信号传导通过抑制Akt / HDM2介导的P53降解和上调P53依赖性DR5表达，诱导肿瘤坏死因子相关诱导配体诱导的人肝细胞癌细胞中的细胞凋亡。

Hypoxia Inhibits Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand-Induced Apoptosis by Blocking Bax Translocation

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