Congestion analysis device, congestion analysis method, and congestion analysis program

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• 3. Congestion degree analysis device, congestion degree analysis system, congestion degree analysis method, and congestion degree analysis program [P] . 日本专利： JP6581787B2 . 2019-09-25

  机译： 拥挤度分析装置，拥挤度分析系统，拥挤度分析方法和拥挤度分析程序

• 4. CONGESTION DEGREE ANALYSIS DEVICE, CONGESTION DEGREE ANALYSIS SYSTEM, CONGESTION DEGREE ANALYSIS METHOD, AND CONGESTION DEGREE ANALYSIS PROGRAM [P] . 日本专利： JP2016177467A . 2016-10-06

  机译： 拥挤度分析设备，拥挤度分析系统，拥挤度分析方法和拥挤度分析程序

• 5. CONGESTION ANALYSIS DEVICE, CONGESTION ANALYSIS METHOD, AND CONGESTION ANALYSIS PROGRAM [P] . 欧洲知识产权局专利： EP3457358A4 . 2019-11-20

  机译： 拥塞分析设备，拥塞分析方法和拥塞分析程序

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. A New Method for Optimal Placement of iTCSC/iBased on Sensitivity Analysis for Congestion Management [J] . Abouzar Samimi ,Peyman Naderi . 智能电网与可再生能源（英文） . 2012,第1期
• 3. TCP/IP拥塞控制机制的显式拥塞指示算法 [C] . 季福坤 . 中国计算机用户协会网络应用分会2008年网络新技术与应用研讨会 . 2008
• 4. Ad Hoc网络中基于拥塞区域的拥塞自适应LAR路由协议研究 [A] . 邓龙 . 2013