METHOD FOR AUTOMATICALLY DETECTING A BASIC POSITION OF A PERSON LYING ON A MATTRESS AND METHOD FOR PRODUCING A GRAPHIC REPRESENTATION

• 1. 用于从在床边或位于某一其他病人位置处的病人获取诊断和预后信息的集成式手持设备及相关的方法 [P] . 中国专利： CN103108592A . 2013-05-15
• 2. 自动产生用于将机器人从停止位置返回预设重启位置的无碰撞返回程序的方法和设备 [P] . 中国专利： CN104812535A . 2015-07-29
• 3. METHOD FOR AUTOMATICALLY DETECTING A BASIC POSITION OF A PERSON LYING ON A MATTRESS AND METHOD FOR PRODUCING A GRAPHIC REPRESENTATION [P] . 世界知识产权组织专利： WO2019072916A1 . 2019-04-18

  机译： 自动检测人在床垫上的基本位置的方法和产生图形表示的方法

• 4. A method of automatically detecting a home position of a person lying on a mattress and generating a graphic representation of that person [P] . AT520485A1 . 2019-04-15

  机译： 一种自动检测躺在床垫上的人的家中位置并生成该人的图形表示的方法

• 5. Method for automatically adjusting hardness of mattress based on operator's lying position or operator's side lying position and device thereof [P] . 美国专利： US8893336B2 . 2014-11-25

  机译： 基于操作员的卧位或侧卧位自动调节床垫硬度的方法及其装置

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• 1. An Automatic Text Region Positioning Method for the Low-Contrast Image [J] . Gongqin Liu ,Murong Jiang ,Helin Cun . 电脑和通信（英文） . 2017,第10期
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• 4. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
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• 6. 基于图形表示方法的蛋白质亚细胞定位预测模型 [A] . 喻道莉 . 2015