METHOD FOR DETERMINING THE PLASTIC CONTENT IN WATER SAMPLES AND IN WASTE WATER SAMPLES

• 1. 一种同时测定水样中7种抗生素含量的方法 [P] . 中国专利： CN113945658A . 2022-01-18
• 2. 中空纤维膜微萃取结合闪蒸气相色谱法测定水样中塑化剂含量的方法 [P] . 中国专利： CN108828099B . 2021.04.06
• 3. Method for determining the plastic content in water and wastewater samples [P] . AT519548A4 . 2018-08-15

  机译： 测定水和废水样品中塑料含量的方法

• 4. Method for determining the plastic content in water and wastewater samples [P] . AT519548B1 . 2018-08-15

  机译： 测定水和废水样品中塑料含量的方法

• 5. Procedure for determining the plastic content in water and wastewater samples [P] . 德国专利： DE112018000603A5 . 2020-01-23

  机译： 测定水和废水样品中塑料含量的程序

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• 1. Optimization Method to Determine Gross Alpha-Beta in Water Samples Using Liquid Scintillation Counter [J] . Waleed M. Abdellah . 水资源与保护（英文） . 2013,第9期
• 2. Optimizing Sampling, Sample Processing and Analysis Methods for Radon (SUP222/SUPRn) in Water by Liquid Scintillation Counting [J] . Uttam K. Saha ,Michael E. Kitto ,Dana R. Lynch . 美国分析化学（英文） . 2018,第1期
• 3. Optimization of Analytical Conditions to Determine Steroids and Pharmaceuticals Drugs in Water Samples Using Solid Phase-Extraction and HPLC [J] . Ramiro Vallejo-Rodriguez ,Alberto Lopez-Lopez ,Hugo Saldarriaga-Norena . 美国分析化学（英文） . 2011,第8期
• 4. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 5. 标准加入法测定模拟海水样品和废水样品中的铜等5种金属元素 [C] . 谭清 ,杨坤红 ,郑疆秋 . 2014中国环境科学学会学术年会 . 2014
• 6. 固定化聚合离子液体分离富集Pb(Ⅱ)及水样中铅含量的测定 [A] . 岳杰 . 2013