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Method of processing data protected against fault injection attacks and associated device

机译：处理防止故障注入攻击的数据的方法及相关设备

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摘要

A method of cryptographic processing of data (X), in particular a method protected against fault injection attacks, and an associated device. The processing includes at least one transformation (100, 100₁-100₆) of an input data item (s) into a result data item (s′). In this case the method includes a step (E204) of verifying the transformation including the following steps:obtaining (E206) a first data item (DV(s′)) that is compressed by applying a compression operation (110, MDV, ADV) to the result data item (s′);obtaining (E208) a second compressed data item (DV(s)) that is compressed by applying the compression operation (110, MDV, ADV) to the input data item (s);determining (E210) a verification data item (DV(s)′) by applying the transformation (100, 100₁-100₆) to the second compressed data item (DV(s)) and;comparing (E212) the verification data item and the first compressed data item.

机译：数据（X）的密码处理方法，特别是防止故障注入攻击的方法，以及相关的设备。该处理包括对输入数据项的至少一个变换（ 100、100 _{1 - 100 _{6 ）（ s）转换为结果数据项（s'）。在这种情况下，该方法包括验证转换的步骤（E 204 ），包括以下步骤：获得（E 206 ）通过应用压缩操作（ 110 ，MDV，ADV）进行压缩的第一数据项（DV（s'）））到结果数据项（s'）; 获得（E 208 ）第二个压缩数据项（DV（ s））通过将压缩操作（ 110 ，MDV，ADV）应用于输入数据项进行压缩; 通过应用变换（ 100，100 _{1 -，确定（E 210 ）验证数据项（DV（s）'） 100 _{6 ）到第二个压缩数据项（DV），并且; 比较（E 212 ）验证数据项和第一个压缩数据项。}}}} 展开▼

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公开/公告号US9559838B2

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公开/公告日2017-01-31

原文格式PDF

申请/专利权人 LAURIE GENELLE;CHRISTOPHE GIRAUD;EMMANUEL PROUFF;
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申请/专利号US20100847156

发明设计人 EMMANUEL PROUFF;LAURIE GENELLE;CHRISTOPHE GIRAUD;
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申请日2010-07-30

分类号G06F21/00;H04L9/06;H04L9/00;

国家 US

入库时间 2022-08-21 13:41:41

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专利

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1. 处理故障注入攻击的安全系统和方法 [P] . 中国专利： CN110048997B . 2021.10.15

2. 处理故障注入攻击的安全系统和方法 [P] . 中国专利： CN110048997A . 2019-07-23

3. METHOD OF PROCESSING DATA PROTECTED AGAINST FAULT INJECTION ATTACKS AND ASSOCIATED DEVICE [P] . 韩国专利： KR101731645B1 . 2017-04-28

机译：处理针对故障注入攻击和相关设备的数据的方法

4. METHOD OF PROCESSING DATA PROTECTED AGAINST FAULT INJECTION ATTACKS AND ASSOCIATED DEVICE [P] . 美国专利： US2011029784A1 . 2011-02-03

机译：处理针对故障注入攻击和相关设备的数据的方法

5. CONDITIONAL DATA PROCESSING METHOD FOR PROTECTING ATTACKS BY GENERATING FAULTS AND DEVICE THEREFOR [P] . 法国专利： FR2934396B1 . 2010-09-17

机译：通过产生故障及其装置来保护攻击的条件数据处理方法

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1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期

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