USER TREMINAL FOR INTERWOKING WITH GATEWAY INSTALLED IN A VEHICLE AND AND METHOD FOR GUIDING A CHANGE OF TRAFFIC SIGNAL USING SMARTPHONE APPLICATION

• 1. 用户终端设备、用于支付的终端、以及使用所述用户终端设备和用于支付的终端进行支付的方法和系统 [P] . 中国专利： CN107667387A . 2018-02-06
• 2. 交互型家电系统、服务器装置、交互型家电设备、用于家电系统进行交互的方法、对用于使计算机实现该方法的程序进行保存的非易失性的计算机可读取数据记录介质 [P] . 中国专利： CN106062869A . 2016-10-26
• 3. USER TREMINAL FOR INTERWOKING WITH GATEWAY INSTALLED IN A VEHICLE AND AND METHOD FOR GUIDING A CHANGE OF TRAFFIC SIGNAL USING SMARTPHONE APPLICATION [P] . KR102310291B1 . 2021-10-07

  机译： 用于使用安装在车辆和方法中的网关互通的用户终端，以及使用智能手机应用引导交通信号的变化

• 4. METHOD FOR SIGNALLING THE APPROACH OF A VEHICLE TO A SMARTPHONE USER AND CORRESPONDING APPLICATION [P] . WO2022008837A1 . 2022-01-13

  机译： 用信号向智能手机用户和相应的应用发送车辆方法的方法

• 5. SYSTEM FOR GUIDING A CHANGE OF TRAFFIC SIGNAL USING GATEWAY AND METHOD THEREOF [P] . 韩国专利： KR20160095861A . 2016-08-12

  机译： 用网关引导交通信号变化的系统及其方法

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期