Device and method to separate a fatty structure connected to a intestine from the intestine by cutting

• 1. 通过切割从肠子分离连接至肠子的脂肪结构的设备和方法 [P] . 中国专利： CN104254250B . 2016.07.20
• 2. 通过切割从肠子分离连接至肠子的脂肪结构的设备和方法 [P] . 中国专利： CN104254250A . 2014-12-31
• 3. DEVICE AND METHOD FOR SEPARATING A FATTY STRUCTURE CONNECTED TO AN INTESTINE FROM THE INTESTINE BY MEANS OF CUTTING [P] . CA2864989C . 2019-12-31

  机译： 通过切割的方法从肠道分离与肠道相连的脂肪结构的装置和方法

• 4. DEVICE AND METHOD FOR SEPARATING A FATTY STRUCTURE CONNECTED TO AN INTESTINE FROM THE INTESTINE BY MEANS OF CUTTING [P] . EA027950B1 . 2017-09-29

  机译： 通过切割的方法从肠道分离与肠道相连的脂肪结构的装置和方法

• 5. DEVICE AND METHOD FOR SEPARATING A FATTY STRUCTURE CONNECTED TO AN INTESTINE FROM THE INTESTINE BY MEANS OF CUTTING [P] . HUE030357T2 . 2017-05-29

  机译： 通过切割的方法从肠道分离与肠道相连的脂肪结构的装置和方法

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• 3. The impact of synbiotic administration through in ovo technology on the microstructure of a broiler chicken small intestine tissue on the 1~（st） and 42~（nd） day of rearing [J] . A.Sobolewska, J.Bogucka, A.Dankowiakowska, 畜牧与生物技术杂志：英文版 . 2018,第1)期
• 4. The impact of synbiotic administration through in ovo technology on the microstructure of a broiler chicken small intestine tissue on the 1st and 42nd day of rearing [J] . A.Sobolewska, J.Bogucka, A.Dankowiakowska, 畜牧与生物技术杂志(英文版) . 2018,第001期
• 5. Effects of goat milk enriched with oligosaccharides on microbiota structures, and correlation between microbiota and short-chain fatty acids in the large intestine of the mouse [J] . Ying Han, Haorui Ma, Yingli Liu, Journal of dairy science . 2021,第3期
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• 11. Lectures to general practitioners on the diseases of the stomach and intestines with an account of their relations to other diseases and of the most recent methods applicable to the diagnosis and treatment of them in general; also "the gastro-intestinal clinic", in which all such diseases are separately considered / [O] . Reed, Boardman,, 1842-1917. 1904

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 丁酸梭菌对鲵鱼肠粘膜结构及肠内短链脂肪酸的影响 [C] . 潘晓东 ,吴天星 ,宋增幅 . 中国畜牧兽医学会动物微生态学分会第四届第十次全国学术研讨会暨动物微生态企业发展论坛 . 2010
• 3. 大鼠肠神经嵴干细胞的分离培养材料的选择及Wnt5a在大鼠肠神经嵴干细胞及其分化过程中的表达 [A] . 牛志新 . 2010