TERMINAL CALLED PROCESSING/TESTING METHOD AND LONG TIME EVOLUTION SYSTEM

• 1. 呼叫处理系统、通信服务器、通信终端及呼叫处理方法 [P] . 中国专利： CN1922804A . 2007-02-28
• 2. 一种聚合物损伤演化测试方法及系统 [P] . 中国专利： CN109187185A . 2019-01-11
• 3. TERMINAL CALLED PROCESSING/TESTING METHOD AND LONG TIME EVOLUTION SYSTEM [P] . 欧洲知识产权局专利： EP2557846B1 . 2016-01-13

  机译： 终端呼叫处理/测试方法和长时间演化系统

• 4. TERMINAL CALLED PROCESSING/TESTING METHOD AND LONG TIME EVOLUTION SYSTEM [P] . 欧洲知识产权局专利： EP2557846A1 . 2013-02-13

  机译： 终端呼叫处理/测试方法和长时间演化系统

• 5. TERMINAL CALLED PROCESSING/TESTING METHOD AND LONG TIME EVOLUTION SYSTEM [P] . 韩国专利： KR20130038846A . 2013-04-18

  机译： 终端呼叫处理/测试方法和长时间演化系统

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• 3. Kovako cement terminal expertiseShipping large loads of any bulk material around the world is no mean feat. Every stage of transport has a cost attached to it so it is in the interest of companies (particularly in these testing times) to use the most effective and efficient methods possible. This article from Kovako describes how being part of FLSmidth has enabled it to deliver comprehensive logistical and handling solutions for the transport of cement. [J] . Global Cement Magazine . 2009,第JulaAuga期
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• 9. Development of a Bio-inspired Vision System for Mobile Micro-robots:In this paper, we present a new bio-inspired vision system for mobile micro-robots. The processing method takes inspiration from vision of locusts in detecting the fast approaching objects. Research suggested that locusts use wide-field visual neuron called the lobula giant movement detector to respond to imminent collisions. We employed the locusts' vision mechanism to motion control of a mobile robot. The selected image processing method is implemented on a developed extension module using a low-cost and fast ARM processor. The vision module is placed on top of a micro-robot to control its trajectory and to avoid obstacles. The observed results from several performed experiments demonstrated that the developed extension module and the inspired vision system are feasible to employ as a vision module for obstacle avoidance and motion control. [O] . Hu, Cheng, Arvin, Farshad, Yue, Shigang 2014
• 10. Test and Evaluation of the Beacon Processing Subsystem of the Automated Radar Terminal System (ARTS III) All-Digital Tracking Level System. [R] . holtz,martin schoenthal,mark -1

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
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