DEVICE OF DIVIDING AND SUPPLYING GAS AND A METHOD OF DIVIDING AND SUPPLYING GAS USING THE SAME, CAPABLE OF PRECISELY DIVIDING AND SUPPLYING GAS

• 1. 从具有流量控制装置的气体供给设备向容器分流地供给气体的气体分流供给装置及气体分流供给方法 [P] . 中国专利： CN100538573C . 2009.09.09
• 2. 从具有流量控制装置的气体供给设备向容器分流地供给气体的气体分流供给装置及气体分流供给方法 [P] . 中国专利： CN1809794A . 2006-07-26
• 3. Apparatus for dividing and supplying gas and method for dividing and supplying gas by use of this apparatus [P] . 美国专利： US9261884B2 . 2016-02-16

  机译： 用于分配和供应气体的设备以及使用该设备分配和供应气体的方法

• 4. Apparatus for dividing and supplying gas and method for dividing and supplying gas [P] . 美国专利： US9477232B2 . 2016-10-25

  机译： 分配和供应气体的装置以及分配和供应气体的方法

• 5. APPARATUS FOR SUPPLYING GAS WHILE DIVIDING AND METHOD FOR SUPPLYING GAS WHILE DIVIDING USING THE SAME [P] . 韩国专利： KR101510146B1 . 2015-04-08

  机译： 用于在分割时供应气体的装置和在使用相同的分割时供应气体的方法

• 1. An optimal method for prediction and adjustment on gasholder level and self-provided power plant gas supply in steel works [J] . LI Hong-juan, WANG Jian-jun, WANG Hua, 中南大学学报（英文版） . 2014,第007期
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• 8. Bridging the operational divide: An information -processing model of internal supply chain integration [D] . Rosado Feger, Ana L. 2009
• 9. Interferon-α blocks HIV-1 infection in non-dividing myeloid cells despite SAMHD1 degradation and high deoxynucleoside triphosphates supply [O] . Loic Dragin, Laura Nguyen, Hichem Lahouassa, 2013
• 10. "A Karnel Regression of Phillips' Data" Abstract: Economists have assumed that the Phillips curve, which shows a positive (negative) relation between inflation and the output ratio (unemployment rate), may be mapped off the aggregate demand -aggregate supply apparatus. The paper shows that the Phillips curve requires that unlikely restrictions be put on the form of the aggregate supply and aggregate demand curves. In this case, it is inappropriate to treat data on inflation and capacity utilization as the basis for estimating an underlying formal model. The paper therefore uses a nonparametric, data-driven method to describe the data. This method, of kernel regression, shows the inflation-unemployment association in Phillips's sample to be negative on a global scale, yet irregular within particular ranges of unemployment. [O] . Nancy J. Wulwick, Y.P. Mack 100

• 1. An optimal method for prediction and adjustment on gasholder level and self-provided power plant gas supply in steel works [J] . 李红娟 ,王建军 ,王华 . 中南大学学报 . 2014,第007期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. 自动气体分割器在气体标准物质分析中的应用 [C] . 李春瑛 ,杜秋芳 . 中国工业气体工业协会2003年年会 . 2003
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