WATER CONTAINMENT APPARATUS AND METHOD, AND ASSOCIATED FASTENER

• 1. 紧固件对准系统、紧固件对准套件和相关方法 [P] . 中国专利： CN110814721A . 2020-02-21
• 2. 用于紧固件设置机的紧固件处理装置及相关方法 [P] . 中国专利： CN111683761A . 2020-09-18
• 3. Water Containment Apparatus and Method, and Associated Fastener [P] . 美国专利： US2015110558A1 . 2015-04-23

  机译： 含水设备和方法，以及相关的紧固件

• 4. WATER CONTAINMENT APPARATUS AND METHOD, AND ASSOCIATED FASTENER [P] . CA2872321A1 . 2013-11-07

  机译： 水资源保护装置和方法，以及相关的紧固件

• 5. Apparatus for filling water in vent pipe of containment vessel and method of testing differential pressure in containment vessel using the apparatus [P] . 日本专利： JP2901729B2 . 1999-06-07

  机译： 用于向安全壳的排气管中注水的设备以及使用该设备测试安全壳中的压差的方法

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• 5. Patent Issued for Fluorescent Agent Concentration Measuring Apparatus, Dose Control Apparatus, Administration System, Fluorescent Agent Concentration Measuring Method,Apparatus, Dose Control Apparatus, Administration System,Fluorescent Agent Concentration Measuring Method, and Dose Control Method [J] . Journal of Engineering . 2013,第13期
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• 1. Study on the treatment of eutrophic water with a new integrative apparatus for water purification [J] . WANG Li-ping ,XUE Chun-yang ,GUO Ying-qing12 . 环境科学与工程 . 2009,第007期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. Different Detection and Treatment Methods for iEntamoeba histolytica/iand iEntamoeba dispar/iin Water/Wastewater: A Review [J] . Rakib Ahmed Chowdhury ,Nwadiuto Esiobu ,Daniel E. Meeroff . 环境保护(英文) . 2022,第1期
• 4. 浅谈汽车紧固件联结知识及常见故障分析方法 [C] . 佘国元 . 第十六届河南省汽车工程科技学术研讨会 . 2020
• 5. 基于Mask R-CNN模型的钢轨紧固件中心定位方法研究 [A] . 杜平 . 2020