METHOD TO MEASURE PARTICLE MOBILITY IN SOLUTION WITH SCATTERED AND UNSCATTERED LIGHT

• 1. 用于散射光测量仪的测试装置，用于散射光测量仪的测试装置的制造方法和用于检验散射光测量仪的方法 [P] . 中国专利： CN105102961B . 2017.12.12
• 2. 用于散射光测量仪的测试装置，用于散射光测量仪的测试装置的制造方法和用于检验散射光测量仪的方法 [P] . 中国专利： CN105102961A . 2015-11-25
• 3. Method to measure particle mobility in solution with scattered and unscattered light [P] . 美国专利： US8525991B2 . 2013-09-03

  机译： 用散射光和非散射光测量溶液中颗粒迁移率的方法

• 4. Apparatus to measure particle mobility in solution with scattered and unscattered light [P] . 美国专利： US8441638B2 . 2013-05-14

  机译： 用散射光和非散射光测量溶液中颗粒迁移率的装置

• 5. Method for determining positions of particles in focused laser beam comprises capturing signals by diode, on which interference pattern is produced by light scattered from particles and unscattered light [P] . 德国专利： DE10130004A1 . 2003-01-09

  机译： 确定聚焦激光束中粒子位置的方法包括通过二极管捕获信号，在该信号上，从粒子散射的光和未散射的光会产生干涉图样

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 利用激光诱导荧光和散射光测量柴油喷雾的油滴直径 [C] . 史绍熙 . 中国内燃机学会第四届学术年会 . 1995
• 3. 荧光和共振瑞利散射光谱法测定某些无机金属离子的新方法研究 [A] . 米莹 . 2016