Display system and method for embeddedly transmitting data signals, control signals, clock signals and setting signals

• 1. 单载波频分多址通信系统中用于传输控制信号和数据信号的频率资源的划分 [P] . 中国专利： CN101682485B . 2013.06.12
• 2. 单载波频分多址通信系统中用于传输控制信号和数据信号的频率资源的划分 [P] . 中国专利： CN101682485A . 2010-03-24
• 3. DISPLAY SYSTEM AND METHOD FOR EMBEDDEDLY TRANSMITTING DATA SIGNALS, CONTROL SIGNALS, CLOCK SIGNALS AND SETTING SIGNALS [P] . 美国专利： US2007171161A1 . 2007-07-26

  机译： 用于嵌入式传输数据信号，控制信号，时钟信号和设置信号的显示系统和方法

• 4. DISPLAY SYSTEM AND METHOD FOR TRANSMITTING DATA SIGNAL, CONTROL SIGNAL, CLOCK SIGNAL AND SETTING SIGNAL WITH EMBEDDING SYSTEM [P] . 日本专利： JP2007193305A . 2007-08-02

  机译： 嵌入式系统传输数据信号，控制信号，时钟信号和设置信号的显示系统和方法

• 5. Display controller, video signal transmitting method and system thereof for transmitting video signals with multiple data rate and reduced numbers of signals line [P] . 美国专利： US9805685B2 . 2017-10-31

  机译： 显示控制器，视频信号传输方法及其系统，用于传输多数据率，信号线数量减少的视频信号

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• 8. Wide Bandwidth - High Accuracy Control Loops in the presence of Slow Varying Signals and Applications in Active Matrix Organic Light Emitting Displays and Sensor Arrays. [D] . Charisoulis, Thomas. 2014
• 9. A mechanical signal transmitted by the flagellum controls signalling in Bacillus subtilis [O] . Lynne S Cairns, Victoria L Marlow, Emma Bissett, -1
• 10. Erratum: Oncogenic KRAS Regulates Tumor Cell Signaling via Stromal Reciprocation:Our paper demonstrated the cell-autonomous and non-cell-autonomous effects of oncogene signaling in tumor and stromal cells using a proteomic approach. It has come to our attention that Data S1, which summarized our proteomic and phosphoproteomic data, included two sets of errors. In the tab related to Figure 3E, the data were labeled as representing log2-transformed ratios but were erroneously formatted to represent natural ratios. These numbers have now been changed to represent log2-transformed ratios. In the tab related to Figure 5, a copying error from our proteomics software caused the 6H time values to be incorrectly displayed. These values have also now been corrected. The values represented in the corrected version of Data S1 were the ones that had been used in our analyses throughout the paper, so the conclusions and figures in the paper remain unchanged. [O] . Tape, Christopher J., Ling, Stephanie, Dimitriadi, Maria, 2016
• 11. System and Method for Adaptively Deskewing Parallel Data Signals Relative to a Clock. [R] . Jenkins, P. N., Cornett, F. N. 2006

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Test for random in electrical signals time series of CO_2 short circuit transition welding process by the method of surrogate data [J] . 王莹 ,吕小青 ,王立君 . 中国焊接：英文版 . 2016,第1期
• 3. 两种非OPC传输RS-485信号实现多系统显示的方法 [C] . 王冰 . 2015中国石油石化仪表与自动化技术及管理交流会 . 2015
• 4. 基于FPGA的无人机自组网视频及控制信号传输系统 [A] . 郭睿达 . 2019