BEACH SAND-CLEANING METHOD AND MECHANISM

• 1. 一种震动筛分式沙滩清理车及其清理方法 [P] . 中国专利： CN109440718B . 2020.06.05
• 2. 一种震动筛分式沙滩清理车及其清理方法 [P] . 中国专利： CN109440718A . 2019-03-08
• 3. BEACH SAND-CLEANING METHOD AND MECHANISM [P] . GR1005509B . 2007-05-08

  机译： 清理沙滩的方法和机理

• 4. METHOD AND MECHANISM FOR CLEANING BEACH SAND [P] . 欧洲知识产权局专利： EP1926860A1 . 2008-06-04

  机译： 清洁沙滩的方法和机理

• 5. METHOD AND MECHANISM FOR CLEANING BEACH SAND [P] . 世界知识产权组织专利： WO2007034245A1 . 2007-03-29

  机译： 清洁沙滩的方法和机理

• 1. Diagenetic Evolution and Formation Mechanisms of High-Quality Reservoirs under Multiple Diagenetic Environmental Constraints: An Example from the Paleogene Beach-Bar Sandstone Reservoirs in the Dongying Depression, Bohai Bay Basin [J] . WANG Jian, CAO Yingchang, SONG Guoqi, 地质学报（英文版） . 2017,第001期
• 2. Preface to the Special Issue on Atmospheric Oxidation Capacity, Ozone, and PM2.5 Pollution: Quantification Methods, Formation Mechanisms, Simulation, and Control [J] . Yuesi WANG, Zirui LIU 大气科学进展（英文版） . 2021,第007期
• 3. Study on the Missing Data Mechanisms and Imputation Methods [J] . Abdullah Z. Alruhaymi, Charles J. Kim 统计学期刊（英文） . 2021,第004期
• 4. SHORING OF LONG BEACH MAIN PUMP STATION UTILIZING GROUND IMPROVEMENT METHODS, LONG BEACH, CA [C] . Gabriel Carvajal, Eric Lindquist, John Compagnone Deep Foundations Institute annual conference on Deep Foundations . 2015
• 5. Computerized approaches to enhance understanding of organic reaction mechanisms: CAN reaction mechanisms and CPLEX prelaboratory methodology. [D] . Al-Shammari, Abdulrahman G. Alhamzani. 2008
• 6. Transcriptome profiling of a beach-adapted wild legume for dissectingnovel mechanisms of salinity tolerance [O] . Hengyou Zhang, Christine Zuelsdorf, Darin Penneys, 2018
• 7. Grand Circulation Process of Beach Cusp and its Seasonal Variation at the Mang-Bang Beach from the Perspective of Trapped Mode Edge Waves as the Driving Mechanism of Beach Cusp Formation [O] . Yong Jun Cho 2019
• 8. Evaluation of Potential Sources and Transport Mechanisms of Fecal Indicator Bacteria to Beach Water, Murphy Park Beach, Door County, Wisconsin. [R] . Juckem, P. F., Corsi, S. R., McDermott, C., 2012

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Examination of Diurnal Variation at a Non-Sewage Impacted Beach via qPCR and Culture Based Methods [J] . Dennis Mudd ,Tamara Anan’eva ,Julie Kinzelman . 环境保护（英文） . 2012,第10期
• 3. 古建筑壁画霉菌病害的特征、机理与清理 [C] . 李峰 . 中国勘察设计协会传统建筑分会彩画油饰专业委员会2017年年会暨中国传统建筑彩画油饰传承与发展学术论坛 . -1
• 4. 沙滩垃圾清理车设计研究 [A] . 刘梦丽 . 2019