Separation mechanism for randomly distributed object

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• 3. Mechanism for the separation of randomly distributed objects (Machine-translation by Google Translate, not legally binding) [P] . ES1058035U . 2004-10-16

  机译： 分离随机分布对象的机制（通过Google Translate进行机器翻译，不具有法律约束力）

• 4. systems and methods for encoding randomly distributed resources on an object [P] . BRPI0500133B1 . 2017-05-23

  机译： 用于编码对象上随机分布的资源的系统和方法

• 5. Preparation of antiforgery label using randomly distributed pattern of the nano and microscale objects formed on various substrates and authenticity verification method [P] . 韩国专利： KR101113035B1 . 2012-02-27

  机译： 利用在各种基材上形成的纳米和微米级物体的随机分布图案制备防伪标签和真实性验证方法

• 1. Analgesic effects and hemodynamic mechanisms of perpendicular and transverse needling at Sanyinjiao(SP 6) in patients with primary dysmenorrhea: A randomized controlled trial [J] . Ali Mohammadi, Mohammad Reza Afshari Fard, Liangxiao Ma, 中医科学杂志(英文) . 2021,第003期
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• 6. The use of liquid chromatography and subcritical fluid chromatography for chiral separations using macrocyclic chiral stationary phases: Insights into mechanisms of retention and chiral discrimination [D] . Woods, Ross Michael. 2014
• 7. Generation Mechanism of Nonlinear Rayleigh Surface Waves for Randomly Distributed Surface Micro-Cracks [O] . Xiangyan Ding, Feilong Li, Youxuan Zhao, 2018
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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 射流床流化分离柱中的颗粒分离—Ⅰ：分离机制与气速选择 [C] . 张济宇 . 第六届全国流态化会议 . 1993
• 3. 随机分布交叉纤维分离算法的研究 [A] . 洪津津 . 2019